Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genome instability independent of type I interferon signaling drives neuropathology caused by impaired ribonucleotide excision repair

18 Oct 2021

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to interferon and immune responses
Share to RNA Research and Splicing
Share to Systemic Lupus Erythematosus Research

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 34655526
DOI: 10.1016/j.neuron.2021.09.040

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genome instability independent of type I interferon signaling drives neuropathology caused by impaired ribonucleotide excision repairPMID 34655526Read ↗

Genome instability and DNA repair in brain, ageing and neurological disease.Semantic proximity Read ↗

Chromatin regulation of DNA damage repair and genome integrity in the central nervous system.Semantic proximity Read ↗

DNA damage in neurodegenerative diseasesSemantic proximity Read ↗

Human nucleotide excision repair syndromes: molecular clues to unexpected intricacies.Semantic proximity Read ↗

Poly (ADP-ribose) polymerase 1 and neurodegenerative diseases: Past, present, and futureSemantic proximity Read ↗

Incorporation of a nucleoside analog maps genome repair sites in post-mitotic human neuronsSemantic proximity Read ↗

DNA damage rather than type I IFN signaling is the primary mediator of neural dysfunction in Aicardi-Goutières syndrome after RNASEH2 disruption.Semantic proximity Read ↗

Poly(ADP-ribose)polymerase-1 hyperactivation in neurodegenerative diseases: The death knell tolls for neuronsSemantic proximity Read ↗

Genome instability and loss of protein homeostasis: converging paths to neurodegeneration?Semantic proximity Read ↗

RNA-mediated toxicity in neurodegenerative diseaseSemantic proximity Read ↗

Activated or Impaired: An Overview of DNA Repair in Neurodegenerative DiseasesSemantic proximity Read ↗

Incorporation of a nucleoside analog maps genome repair sites in postmitotic human neurons.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.