Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Vimentin Promotes the Aggressiveness of Triple Negative Breast Cancer Cells Surviving Chemotherapeutic Treatment

15 Jun 2021

Abstract excerpt

Tremendous data have been accumulated in the effort to understand chemoresistance of triple negative breast cancer (TNBC). However, modifications in cancer cells surviving combined and sequential treatment still remain poorly described. In order to mimic clinical neoadjuvant treatment, we first treated MDA-MB-231 and SUM159-PT TNBC cell lines with epirubicin and cyclophosphamide for 2 days, and then with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to HER2/EGFR in Cancer Research
Share to Silk-based biomaterials and applications
Share to Skin and Cellular Biology Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34203746
DOI: 10.3390/cells10061504

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Vimentin Promotes the Aggressiveness of Triple Negative Breast Cancer Cells Surviving Chemotherapeutic TreatmentPMID 34203746Read ↗

Doxorubicin-Resistant TNBC Cells Exhibit Rapid Growth with Cancer Stem Cell-like Properties and EMT Phenotype, Which Can Be Transferred to Parental Cells through Autocrine SignalingSemantic proximity Read ↗

Molecular heterogeneity in breast carcinoma cells with increased invasive capacities.Semantic proximity Read ↗

Phenotypic Heterogeneity of Triple-Negative Breast Cancer Mediated by Epithelial–Mesenchymal PlasticitySemantic proximity Read ↗

Post-EMT: Cadherin-11 mediates cancer hijacking fibroblastsSemantic proximity Read ↗

Vimentin Is at the Heart of Epithelial Mesenchymal Transition (EMT) Mediated MetastasisSemantic proximity Read ↗

Integrins Alpha-2 and Beta-1 expression increases through Multiple Generations of the EDW01 Patient-Derived Xenograft Model of Breast Cancer - insight into their role in Epithelial Mesenchymal Transition in vivo gained from an in vitro model systemSemantic proximity Read ↗

Triple-negative breast cancer metastasis involves complex epithelial-mesenchymal transition dynamics and requires vimentinSemantic proximity Read ↗

Recurring cycles of deprivation of serum and migration in confined spaces augments ganglioside SSEA-4 expression, boosting clonogenicity and cisplatin resistance in TNBC cell lineSemantic proximity Read ↗

Molecular mechanism of vimentin nuclear localization associated with the migration and invasion of daughter cells derived from polyploid giant cancer cellsSemantic proximity Read ↗

Lovastatin Inhibits EMT and Metastasis of Triple-Negative Breast Cancer Stem Cells Through Dysregulation of Cytoskeleton-Associated ProteinsSemantic proximity Read ↗

Recurring Cycles of Deprivation of Serum and Migration in Confined Spaces Augments Ganglioside SSEA-4 Expression, Boosting Clonogenicity andCisplatin Resistance in TNBC Cell LineSemantic proximity Read ↗

Comparative Analysis of the Development of Acquired Radioresistance in Canine and Human Mammary Cancer Cell LinesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.