Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The Innate Immune Glycoprotein Lactoferrin Represses the Helicobacter pylori cag Type IV Secretion System

25 Jun 2021

Abstract excerpt

Chronic infection with Helicobacter pylori increases risk of gastric diseases including gastric cancer. Despite development of a robust immune response, H. pylori persists in the gastric niche. Progression of gastric inflammation to serious disease outcomes is associated with infection with H. pylori strains which encode the cag Type IV Secretion System (cag T4SS). The cag T4SS is responsible for translocating...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Galectins and Cancer Biology
Share to Gastrointestinal disorders and treatments
Share to Helicobacter pylori-related gastroenterology studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34169626
DOI: 10.1002/cbic.202100249

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The Innate Immune Glycoprotein Lactoferrin Represses the Helicobacter pylori cag Type IV Secretion SystemPMID 34169626Read ↗

Composition, Structure and Function of the Helicobacter Pylori cag Pathogenicity Island Encoded type IV Secretion SystemSemantic proximity Read ↗

Helicobacter pylori Exploits a Unique Repertoire of Type IV Secretion System Components for Pilus Assembly at the Bacteria-Host Cell InterfaceSemantic proximity Read ↗

Pathogenesis of <i>Helicobacter pylori</i> InfectionSemantic proximity Read ↗

Pathogenicity and virulence of Helicobacter pylori : A paradigm of chronic infectionSemantic proximity Read ↗

Helicobacter pylori CagA and Cag type IV secretion system activity have key roles in triggering gastric transcriptional and proteomic alterationsSemantic proximity Read ↗

Roles of the components of the cag -pathogenicity island encoded type IV secretion system in Helicobacter pyloriSemantic proximity Read ↗

Unraveling Helicobacter pylori: Insights into Pathogenesis, Immune Evasion, and Progress Toward Effective VaccinationSemantic proximity Read ↗

Helicobacter pylori γ-glutamyltransferase is linked to proteomic adaptions important for colonizationSemantic proximity Read ↗

Helicobacter pylori infection perturbs iron homeostasis in gastric epithelial cells.Semantic proximity Read ↗

Immune evasion strategies used by Helicobacter pylori.Semantic proximity Read ↗

Helicobacter pylori regulates pro- and anti-inflammatory macrophage responses to promote bacterial survivalSemantic proximity Read ↗

Macrophages recognize the Helicobacter pylori type IV secretion system in the absence of toll-like receptor signallingSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.