Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Temozolomide Treatment Increases Fatty Acid Uptake in Glioblastoma Stem Cells

26 Oct 2020

Abstract excerpt

Among all cancers, glioblastoma (GBM) remains one of the least treatable. One key factor in this resistance is a subpopulation of tumor cells termed glioma stem cells (GSCs). These cells are highly resistant to current treatment modalities, possess marked self-renewal capacity, and are considered key drivers of tumor recurrence. Further complicating an understanding of GBM, evidence shows that the GSC population...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer, Lipids, and Metabolism
Share to Cancer, Hypoxia, and Metabolism
Share to Glioma Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33114573
DOI: 10.3390/cancers12113126

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Temozolomide Treatment Increases Fatty Acid Uptake in Glioblastoma Stem CellsPMID 33114573Read ↗

Glioblastoma Metabolism: Insights and Therapeutic StrategiesSemantic proximity Read ↗

Glioblastoma Stem-Like Cells, Metabolic Strategy to Kill a Challenging TargetSemantic proximity Read ↗

Single-cell and spatial transcriptomic insights into glioma cellular heterogeneity and metabolic adaptationsSemantic proximity Read ↗

Enhanced fatty acid oxidation provides glioblastoma cells metabolic plasticity to accommodate to its dynamic nutrient microenvironment.Semantic proximity Read ↗

Altered lipid metabolism marks glioblastoma stem and non-stem cells in separate tumor nichesSemantic proximity Read ↗

Metabolic and epigenetic reprogramming in the pathogenesis of glioblastoma: Toward the establishment of “metabolism‐based pathology”Semantic proximity Read ↗

Metabolism and signaling crosstalk in glioblastoma progression and therapy resistanceSemantic proximity Read ↗

Advancing glioblastoma treatment by targeting metabolismSemantic proximity Read ↗

The Quiescent Metabolic Phenotype of Glioma Stem CellsSemantic proximity Read ↗

Glioblastoma gene expression based subtypes have defined metabolomic statesSemantic proximity Read ↗

Glioblastomas: Hijacking Metabolism to Build a Flexible Shield for Therapy ResistanceSemantic proximity Read ↗

Glioma Stem-Like Cells and Metabolism: Potential for Novel Therapeutic StrategiesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.