Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Non-coding structural variation differentially impacts attention-deficit hyperactivity disorder (ADHD) gene networks in African American vs Caucasian children.

Scientific reports - 17 Sept 2020

Liu Yichuan, Chang Xiao, Qu Huiqi, Glessner Joseph, Tian Lifeng, Li Dong, Qiu Haijun, Sleiman Patrick M A, Hakonarson Hakon

Abstract excerpt

Previous studies of attention-deficit hyperactivity disorder (ADHD) have suggested that structural variants (SVs) play an important role but these were mainly studied in subjects of European ancestry and focused on coding regions. In this study, we sought to address the role of SVs in non-European populations and outside of coding regions. To that end, we generated whole genome sequence (WGS) data on 875...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Attention Deficit Disorder with Hyperactivity
Share to Black or African American

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Case-Control Studies

Share to Gene Regulatory Networks

Share to RNA, Untranslated

Share to White People

Share to Whole Genome Sequencing

Source

Open in PubMed Open full text

Identifiers

PMID: 32943653
DOI: 10.1038/s41598-020-71307-0
PMCID: PMC7499198

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Non-coding structural variation differentially impacts attention-deficit hyperactivity disorder (ADHD) gene networks in African American vs Caucasian children.PMID 32943653Read ↗

Rare Recurrent Variants in Noncoding Regions Impact Attention-Deficit Hyperactivity Disorder (ADHD) Gene Networks in Children of both African American and European American Ancestry.Semantic proximity Read ↗

Discovery of the first genome-wide significant risk loci for ADHDSemantic proximity Read ↗

Insights into attention-deficit/hyperactivity disorder from recent genetic studies.Semantic proximity Read ↗

The molecular genetic architecture of attention deficit hyperactivity disorder.Semantic proximity Read ↗

Recent developments in the genetics of attention-deficit hyperactivity disorder.Semantic proximity Read ↗

An integrative approach to investigate the respective roles of single-nucleotide variants and copy-number variants in Attention-Deficit/Hyperactivity Disorder.Semantic proximity Read ↗

Rare genetic variants confer a high risk of ADHD and implicate neuronal biology.Semantic proximity Read ↗

Attention-deficit/hyperactivity disorder genomics: update for clinicians.Semantic proximity Read ↗

Genome-wide analyses of ADHD identify 27 risk loci, refine the genetic architecture and implicate several cognitive domains.Semantic proximity Read ↗

Genetic Insights Into ADHD BiologySemantic proximity Read ↗

Rare structural variants found in attention-deficit hyperactivity disorder are preferentially associated with neurodevelopmental genes.Semantic proximity Read ↗

Genetics of attention-deficit/hyperactivity disorder: an update.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.