Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cancer Stem-Cell Marker CD44v9-Positive Cells Arise From Helicobacter pylori–Infected CAPZA1-Overexpressing Cells

1 Jan 2019

Abstract excerpt

BACKGROUND & AIMS: CD44 variant 9 (CD44v9)-positive cancer stem-like cells strongly contribute to the development and recurrence of gastric cancer. However, the origin of CD44v9-positive cells is uncertain. METHODS: CD44v9, β-catenin, and epithelial splicing regulatory protein 1 signals were assessed by real-time reverse-transcription polymerase chain reaction, immunoblot analysis, or immunofluorescence...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Helicobacter pylori-related gastroenterology studies
Share to Proteoglycans and glycosaminoglycans research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31146068
DOI: 10.1016/j.jcmgh.2019.05.008

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cancer Stem-Cell Marker CD44v9-Positive Cells Arise From Helicobacter pylori–Infected CAPZA1-Overexpressing CellsPMID 31146068Read ↗

Initiation and Maintenance of Gastric Cancer: A Focus on CD44 Variant Isoforms and Cancer Stem CellsSemantic proximity Read ↗

Mechanisms of the Epithelial–Mesenchymal Transition and Tumor Microenvironment in Helicobacter pylori-Induced Gastric CancerSemantic proximity Read ↗

circFAM73A Promotes Cancer Stem Cell-like Properties of Gastric Cancer Through miR-490-3p/HMGA2 Positive Feedback and HNRNPK-mediated β-catenin Stabilization.Semantic proximity Read ↗

More powerful dysregulation of Helicobacter pylori East Asian-type CagA on intracellular signalingsSemantic proximity Read ↗

TM4SF1-AS1 inhibits apoptosis by promoting stress granule formation in cancer cellsSemantic proximity Read ↗

CD44v8-10 Is a Cancer-Specific Marker for Gastric Cancer Stem CellsSemantic proximity Read ↗

SOX9 Elevation Acts with Canonical WNT Signaling to Drive Gastric Cancer ProgressionSemantic proximity Read ↗

Identification of Key Genes for Controlling Cancer Stem Cell Characteristics in Gastric CancerSemantic proximity Read ↗

Dysregulation of stem cell signaling network due to germline mutation, SNP, Helicobacter pylori infection, epigenetic change and genetic alteration in gastric cancer.Semantic proximity Read ↗

Helicobacter pylori cancer associated CagA protein drives intestinal metaplastic transition in human gastric organoidsSemantic proximity Read ↗

Comprehensive analysis of transcriptome data stemness indices identifies key genes for controlling cancer stem cell characteristics in gastric cancerSemantic proximity Read ↗

Loss of ATOH1 in Pit Cell Drives Stemness and Progression of Gastric Adenocarcinoma by Activating AKT/mTOR Signaling through GAS1Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.