Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Loss of exosomal miR-3188 in cancer-associated fibroblasts contributes to HNC progression

8 Apr 2019

Abstract excerpt

BACKGROUND: Head and neck cancer (HNC) is one of the most common deadly diseases worldwide. An increasing number of studies have recently focused on the malignant functions of cancer-associated fibroblasts (CAFs) in numerous cancers. However, the underlying mechanisms by which CAF-derived exosomes promote tumor progression need to be further elucidated. This study aims to determine whether the loss of specific...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Extracellular vesicles in disease
Share to MicroRNA in disease regulation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30961650
DOI: 10.1186/s13046-019-1144-9

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Loss of exosomal miR-3188 in cancer-associated fibroblasts contributes to HNC progressionPMID 30961650Read ↗

Loss of exosomal miR-34c-5p in cancer-associated fibroblast for the maintenance of stem-like phenotypes of laryngeal cancer cells.Semantic proximity Read ↗

Aberrant expression profiles and bioinformatic analysis of CAF-derived exosomal miRNAs from three moderately differentiated supraglottic LSCC patientsSemantic proximity Read ↗

Exosomal miR-3180-3p Inhibits Proliferation and Metastasis of Non-Small Cell Lung Cancer by Downregulating FOXP4Semantic proximity Read ↗

Suppression of Lymphocyte Functions by Plasma Exosomes Correlates with Disease Activity in Patients with Head and Neck CancerSemantic proximity Read ↗

Transcriptome reprogramming by cancer exosomes: identification of novel molecular targets in matrix and immune modulationSemantic proximity Read ↗

Potential oncogenic roles of mutant-p53-derived exosomes in the tumor-host interaction of head and neck cancers.Semantic proximity Read ↗

Analysis of mRNA-miRNA interaction network reveals the role of CAFs-derived exosomes in the immune regulation of oral squamous cell carcinomaSemantic proximity Read ↗

Tumor-derived exosomal miR-103a-2-5p facilitates esophageal squamous cell carcinoma cell proliferation and migration.Semantic proximity Read ↗

M2 macrophage-derived exosomal EHF transcriptionally activates FGFR1 to promote malignant phenotypes and glycolysis in NSCLC.Semantic proximity Read ↗

Exosomes: a new perspective in EGFR-mutated lung cancer.Semantic proximity Read ↗

Tumor-derived exosomes influence the cell cycle and cell migration of human esophageal cancer cell lines.Semantic proximity Read ↗

Annexin A1 regulates EGFR activity and alters EGFR-containing tumour-derived exosomes in head and neck cancers.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.