Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Nine genes abundantly expressed in the epididymis are not essential for male fecundity in mice

29 Mar 2019

Abstract excerpt

BACKGROUND: Spermatozoa become competent for fertilization during transit through the epididymis. As spermatozoa from the proximal caudal epididymis can fertilize eggs, proteins from the caput and corpus epididymis are required for sperm maturation. OBJECTIVES: Microarray analysis identified that more than 17,000 genes are expressed in the epididymis; however, few of these genes demonstrate expression restricted...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities
Share to Reproductive Biology and Fertility
Share to Sperm and Testicular Function

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30927342
DOI: 10.1111/andr.12621

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Nine genes abundantly expressed in the epididymis are not essential for male fecundity in micePMID 30927342Read ↗

CRISPR/Cas9-mediated genome editing reveals 12 testis-enriched genes dispensable for male fertility in miceSemantic proximity Read ↗

Large-scale discovery of male reproductive tract-specific genes through analysis of RNA-seq datasetsSemantic proximity Read ↗

CRISPR/Cas9-based genome editing in mice uncovers 13 testis- or epididymis-enriched genes individually dispensable for male reproduction†Semantic proximity Read ↗

A novel mouse line with epididymal initial segment-specific expression of Cre recombinase driven by the endogenous Lcn9 promoter.Semantic proximity Read ↗

Individual disruption of 12 testis-enriched genes via the CRISPR/Cas9 system does not affect the fertility of male miceSemantic proximity Read ↗

CRISPR/Cas9-mediated genome editing reveals 30 testis-enriched genes dispensable for male fertility in mice†Semantic proximity Read ↗

<i>Tex11</i> Mutant Mouse Models of Human AzoospermiaSemantic proximity Read ↗

Genome engineering uncovers 54 evolutionarily conserved and testis-enriched genes that are not required for male fertility in miceSemantic proximity Read ↗

Discovery in Silico and Characterization in Vitro of Novel Genes Exclusively Expressed in the Mouse EpididymisSemantic proximity Read ↗

Gene and protein expression in the epididymis of infertile c-ros receptor tyrosine kinase-deficient mice.Semantic proximity Read ↗

Sperm Proteome Maturation in the Mouse Epididymis.Semantic proximity Read ↗

Epididymal cysteine-rich secretory proteins are required for epididymal sperm maturation and optimal sperm functionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.