Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic background may contribute to the latitude-dependent prevalence of dermatomyositis and anti-TIF1-γ autoantibodies in adult patients with myositis.

Arthritis research & therapy - 8 Jun 2018

Parkes Joanna E, Rothwell Simon, Oldroyd Alexander, Chinoy Hector, Lamb Janine A

Abstract excerpt

BACKGROUND: The prevalence of dermatomyositis (DM) versus DM and polymyositis (PM) combined has been shown to be negatively associated with latitude. This observation has been attributed to increasing exposure to ultraviolet (UV) light towards the equator. In this study, we investigated whether differing genetic background in populations could contribute to this distribution of DM. METHODS: Case data derived from...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoantibodies
Share to Dermatomyositis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Background

Share to Inflammatory Myopathies and Dermatomyositis

Share to Polymorphism, Single Nucleotide

Share to Prevalence

Share to Sunlight

Share to Transcription Factors

Source

Open in PubMed Open full text

Identifiers

PMID: 29884237
DOI: 10.1186/s13075-018-1617-9
PMCID: PMC5994128

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic background may contribute to the latitude-dependent prevalence of dermatomyositis and anti-TIF1-γ autoantibodies in adult patients with myositis.PMID 29884237Read ↗

Dense genotyping of immune-related loci in idiopathic inflammatory myopathies confirms HLA alleles as the strongest genetic risk factor and suggests different genetic background for major clinical subgroups.Semantic proximity Read ↗

Global surface ultraviolet radiation intensity may modulate the clinical and immunologic expression of autoimmune muscle disease.Semantic proximity Read ↗

The Genetics of Autoimmune MyositisSemantic proximity Read ↗

Strategies for evaluating idiopathic inflammatory myopathy disease susceptibility genes.Semantic proximity Read ↗

Brief Report: Ultraviolet Radiation Exposure Is Associated With Clinical and Autoantibody Phenotypes in Juvenile MyositisSemantic proximity Read ↗

Identification of genetic variants associated with idiopathic inflammatory myopathies via cross-trait analysis with B cell lymphomasSemantic proximity Read ↗

Identification of Novel Associations and Localization of Signals in Idiopathic Inflammatory Myopathies Using Genome-Wide Imputation.Semantic proximity Read ↗

Ultraviolet radiation intensity predicts the relative distribution of dermatomyositis and anti-Mi-2 autoantibodies in women.Semantic proximity Read ↗

Genetic risk and protective factors for the idiopathic inflammatory myopathies.Semantic proximity Read ↗

Association between IL1B rs16944 polymorphism and the risk of idiopathic inflammatory myopathies.Semantic proximity Read ↗

Immunoglobulin gene polymorphisms are susceptibility factors in clinical and autoantibody subgroups of the idiopathic inflammatory myopathies.Semantic proximity Read ↗

Recent advances in the immunogenetics of idiopathic inflammatory myopathy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.