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Article

ASF1a Promotes Non-homologous End Joining Repair by Facilitating Phosphorylation of MDC1 by ATM at Double-Strand Breaks

21 Sept 2017

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 28943310
DOI: 10.1016/j.molcel.2017.08.021

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

ASF1a Promotes Non-homologous End Joining Repair by Facilitating Phosphorylation of MDC1 by ATM at Double-Strand BreaksPMID 28943310Read ↗

The ACF1 Complex Is Required for DNA Double-Strand Break Repair in Human CellsSemantic proximity Read ↗

ATM and the Mre11 complex combine to recognize and signal DNA double-strand breaksSemantic proximity Read ↗

Histone chaperone ASF1 acts with RIF1 to promote DNA end joining in BRCA1-deficient cellsSemantic proximity Read ↗

Repair of DNA double-strand breaks by mammalian alternative end-joining pathwaysSemantic proximity Read ↗

APE1 senses DNA single-strand breaks for repair and signalingSemantic proximity Read ↗

Human RAP1 inhibits non‐homologous end joining at telomeresSemantic proximity Read ↗

The Interaction of CtIP and Nbs1 Connects CDK and ATM to Regulate HR–Mediated Double-Strand Break RepairSemantic proximity Read ↗

The BAP1 nuclear deubiquitinase is involved in the nonhomologous end-joining pathway of double-strand DNA repair through interaction with DNA-PKSemantic proximity Read ↗

Remodeling and spacing factor 1 (RSF1) deposits centromere proteins at DNA double-strand breaks to promote non-homologous end-joiningSemantic proximity Read ↗

Investigating the regulation of DNA non-homologous end-joining through Ku70/80 interacting factorsSemantic proximity Read ↗

APE2 promotes DNA damage response pathway from a single-strand breakSemantic proximity Read ↗

ATR and ATM differently regulate WRN to prevent DSBs at stalled replication forks and promote replication fork recoverySemantic proximity Read ↗

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