Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Manipulation of the Growth Hormone-Insulin-Like Growth Factor (GH-IGF) Axis: A Treatment Strategy to Reverse the Effects of Early Life Developmental Programming

8 Aug 2017

Abstract excerpt

Evidence from human clinical, epidemiological, and experimental animal models has clearly highlighted a link between the early life environment and an increased risk for a range of cardiometabolic disorders in later life. In particular, altered maternal nutrition, including both undernutrition and overnutrition, spanning exposure windows that cover the period from preconception through to early infancy, clearly...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Diet and metabolism studies
Share to Growth Hormone and Insulin-like Growth Factors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 28786951
DOI: 10.3390/ijms18081729

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Manipulation of the Growth Hormone-Insulin-Like Growth Factor (GH-IGF) Axis: A Treatment Strategy to Reverse the Effects of Early Life Developmental ProgrammingPMID 28786951Read ↗

Developmental origins of the metabolic syndrome: prediction, plasticity, and programming.Semantic proximity Read ↗

Experimental Models of Maternal Obesity and Neuroendocrine Programming of Metabolic Disorders in OffspringSemantic proximity Read ↗

Developmental programming of health and diseaseSemantic proximity Read ↗

Mechanisms by which poor early growth programs type-2 diabetes, obesity and the metabolic syndrome.Semantic proximity Read ↗

Developmental origins of health and disease: brief history of the approach and current focus on epigenetic mechanisms.Semantic proximity Read ↗

Prenatal Programming and Epigenetics in the Genesis of the Cardiorenal SyndromeSemantic proximity Read ↗

Early life programming of obesity and metabolic disease.Semantic proximity Read ↗

Developmental programming of energy balance and the metabolic syndrome.Semantic proximity Read ↗

Fetal Programming of Body Composition, Obesity, and Metabolic Function: The Role of Intrauterine Stress and Stress BiologySemantic proximity Read ↗

Maternal obesity, diabetes during pregnancy and epigenetic mechanisms that influence the developmental origins of cardiometabolic disease in the offspringSemantic proximity Read ↗

Genetic and epigenetic catalysts in early-life programming of adult cardiometabolic disordersSemantic proximity Read ↗

Fetal Growth and Intrauterine Epigenetic Programming of Obesity and Cardiometabolic DiseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.