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Article

USP39, a direct target of microRNA-133a, promotes progression of pancreatic cancer via the AKT pathway

9 Mar 2017

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 28286270
DOI: 10.1016/j.bbrc.2017.03.025

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Related research

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Click a neighbor to travelStep 1 · 12 closest

USP39, a direct target of microRNA-133a, promotes progression of pancreatic cancer via the AKT pathwayPMID 28286270Read ↗

The USP22 promotes the growth of cancer cells through the DYRK1A in pancreatic ductal adenocarcinomaSemantic proximity Read ↗

USP33 promotes pancreatic cancer malignant phenotype through the regulation of TGFBR2/TGFβ signaling pathway.Semantic proximity Read ↗

Role of the USP family in autophagy regulation and cancer progressionSemantic proximity Read ↗

USP19 potentiates autophagic cell death via inhibiting mTOR pathway through deubiquitinating NEK9 in pancreatic cancerSemantic proximity Read ↗

USP13 functions as a tumor suppressor by blocking the NF-kB-mediated PTEN downregulation in human bladder cancerSemantic proximity Read ↗

Tumor Suppressor Functions of miRNA-375 in Nasopharyngeal Carcinoma through Inhibition of Ubiquitin-Specific Protease 1 ExpressionSemantic proximity Read ↗

USP18 promotes cell proliferation and suppressed apoptosis in cervical cancer cells via activating AKT signaling pathwaySemantic proximity Read ↗

USP39 promotes colorectal cancer growth and metastasis through the Wnt/β-catenin pathwaySemantic proximity Read ↗

Ubiquitin specific peptidase 5 enhances STAT3 signaling and promotes migration and invasion in Pancreatic CancerSemantic proximity Read ↗

Involvement of CD40 Targeting miR-224 and miR-486 on the Progression of Pancreatic Ductal AdenocarcinomasSemantic proximity Read ↗

Deubiquitinase USP18 promotes the progression of pancreatic cancer via enhancing the Notch1-c-Myc axisSemantic proximity Read ↗

USP5 promotes tumorigenesis and progression of pancreatic cancer by stabilizing FoxM1 proteinSemantic proximity Read ↗

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