Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Systemic Analysis of Atg5-Null Mice Rescued from Neonatal Lethality by Transgenic ATG5 Expression in Neurons

1 Oct 2016

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Advanced Glycation End Products research
Share to Autophagy in Disease and Therapy
Share to Neuroinflammation and Neurodegeneration Mechanisms

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 27693508
DOI: 10.1016/j.devcel.2016.09.001

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Systemic Analysis of Atg5-Null Mice Rescued from Neonatal Lethality by Transgenic ATG5 Expression in NeuronsPMID 27693508Read ↗

Characterization of a novel neurodevelopmental rare disease caused by a mutation within the autophagy gene <i>ATG9B</i>Semantic proximity Read ↗

Role of<i><i>Epg5</i></i>in selective neurodegeneration and Vici syndromeSemantic proximity Read ↗

Bi-allelic ATG12 variants impair autophagy and cause a neurodevelopmental disorder.Semantic proximity Read ↗

A novel Atg5-shRNA mouse model enables temporal control of Autophagy in vivo.Semantic proximity Read ↗

Overexpression of Atg5 in mice activates autophagy and extends lifespanSemantic proximity Read ↗

EPG5-related Vici syndrome: a paradigm of neurodevelopmental disorders with defective autophagy.Semantic proximity Read ↗

Mutation in ATG5 reduces autophagy and leads to ataxia with developmental delay.Semantic proximity Read ↗

Bi-allelic <i>ATG12</i> variants impair autophagy and cause a neurodevelopmental disorderSemantic proximity Read ↗

Knockout of Atg5 delays the maturation and reduces the survival of adult-generated neurons in the hippocampusSemantic proximity Read ↗

Biallelic ATG9B Variants Define a Novel Autophagy-Related Neurodevelopmental Disorder with Cerebellar Ataxia.Semantic proximity Read ↗

Autophagy‐related gene Atg5 is essential for astrocyte differentiation in the developing mouse cortexSemantic proximity Read ↗

Developmental Consequences of Defective ATG7-Mediated Autophagy in Humans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.