Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enhanced HDL Functionality in Small HDL Species Produced Upon Remodeling of HDL by Reconstituted HDL, CSL112

20 Jul 2016

Abstract excerpt

RATIONALE: CSL112, human apolipoprotein A-I (apoA-I) reconstituted with phosphatidylcholine, is known to cause a dramatic rise in small high-density lipoprotein (HDL). OBJECTIVE: To explore the mechanisms by which the formation of small HDL particles is induced by CSL112. METHODS AND RESULTS: Infusion of CSL112 into humans caused elevation of 2 small diameter HDL fractions and 1 large diameter fraction. Ex vivo...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cholesterol and Lipid Metabolism
Share to Diabetes, Cardiovascular Risks, and Lipoproteins
Share to Lipoproteins and Cardiovascular Health

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 27436846
DOI: 10.1161/circresaha.116.308685

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enhanced HDL Functionality in Small HDL Species Produced Upon Remodeling of HDL by Reconstituted HDL, CSL112PMID 27436846Read ↗

HDLs in crisesSemantic proximity Read ↗

Modifications of the 22A apoA-I mimetic peptide sequence improve the anti-atherosclerotic properties of synthetic HDLSemantic proximity Read ↗

HDL, Atherosclerosis, and Emerging TherapiesSemantic proximity Read ↗

HDL in the 21st Century: A Multifunctional Roadmap for Future HDL ResearchSemantic proximity Read ↗

Role of apoA-I, ABCA1, LCAT, and SR-BI in the biogenesis of HDL.Semantic proximity Read ↗

Distinct roles of size-defined HDL subpopulations in cardiovascular diseaseSemantic proximity Read ↗

Quo Vadis after AEGIS: New Opportunities for Therapies Targeted at Reverse Cholesterol Transport?Semantic proximity Read ↗

A thumbwheel mechanism for APOA1 activation of LCAT activity in HDL.Semantic proximity Read ↗

HDL Dysfunction Caused by Mutations in apoA-I and Other Genes that are Critical for HDL Biogenesis and Remodeling.Semantic proximity Read ↗

HDL biogenesis and functions: Role of HDL quality and quantity in atherosclerosisSemantic proximity Read ↗

Phospholipidation of HDL--how much is too much?Semantic proximity Read ↗

Demystifying HDL Cholesterol-A "Human Knockout" to the Rescue?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.