Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mesenchymal-Epithelial Transition in Sarcomas Is Controlled by the Combinatorial Expression of MicroRNA 200s and GRHL2

12 Jul 2016

Abstract excerpt

Phenotypic plasticity involves a process in which cells transiently acquire phenotypic traits of another lineage. Two commonly studied types of phenotypic plasticity are epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET). In carcinomas, EMT drives invasion and metastatic dissemination, while MET is proposed to play a role in metastatic colonization. Phenotypic plasticity in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to MicroRNA in disease regulation
Share to Sarcoma Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 27402864
DOI: 10.1128/mcb.00373-16

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mesenchymal-Epithelial Transition in Sarcomas Is Controlled by the Combinatorial Expression of MicroRNA 200s and GRHL2PMID 27402864Read ↗

Epithelial-mesenchymal plasticity: How have quantitative mathematical models helped improve our understanding?Semantic proximity Read ↗

Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?Semantic proximity Read ↗

Molecular mechanisms underpinning sarcomas and implications for current and future therapySemantic proximity Read ↗

Stability of the hybrid epithelial/mesenchymal phenotype.Semantic proximity Read ↗

Mechanisms of Disease: epithelial–mesenchymal transition—does cellular plasticity fuel neoplastic progression?Semantic proximity Read ↗

Hybrid E/M Phenotype(s) and Stemness: A Mechanistic Connection Embedded in Network TopologySemantic proximity Read ↗

Hybrid E/M phenotype(s) and stemness: a mechanistic connection embedded in network topologySemantic proximity Read ↗

Epithelial-Mesenchymal Transition in Cancer: Parallels Between Normal Development and Tumor ProgressionSemantic proximity Read ↗

From shape-shifting embryonic cells to oncology: The fascinating history of epithelial mesenchymal transitionSemantic proximity Read ↗

Characteristics and transcriptional regulators of spontaneous epithelial–mesenchymal transition in real-world genetically unperturbed non-spindle breast carcinomaSemantic proximity Read ↗

Epithelial–mesenchymal plasticity in cancer: signaling pathways and therapeutic targetsSemantic proximity Read ↗

ZEB/miR‐200 feedback loop: At the crossroads of signal transduction in cancerSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.