Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

α-Synuclein-induced myelination deficit defines a novel interventional target for multiple system atrophy

8 Apr 2016

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Nerve injury and regeneration
Share to Nuclear Receptors and Signaling
Share to Parkinson's Disease Mechanisms and Treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 27059609
DOI: 10.1007/s00401-016-1572-y

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

α-Synuclein-induced myelination deficit defines a novel interventional target for multiple system atrophyPMID 27059609Read ↗

α-Synuclein impairs oligodendrocyte progenitor maturation in multiple system atrophySemantic proximity Read ↗

Mouse Model of Multiple System Atrophy α-Synuclein Expression in Oligodendrocytes Causes Glial and Neuronal DegenerationSemantic proximity Read ↗

Targeting α-synuclein for treatment of Parkinson's disease: mechanistic and therapeutic considerationsSemantic proximity Read ↗

Multiple-system atrophy: a new α-synuclein disease?Semantic proximity Read ↗

α-synuclein aggregation reduces nigral myocyte enhancer Factor-2D in idiopathic and experimental Parkinson's diseaseSemantic proximity Read ↗

Multiple System Atrophy: Pathology, Pathogenesis, and Path ForwardSemantic proximity Read ↗

Mitochondrial alpha-synuclein accumulation impairs complex I function in dopaminergic neurons and results in increased mitophagy in vivoSemantic proximity Read ↗

The role of ?-synuclein in the pathogenesis of multiple system atrophySemantic proximity Read ↗

Pathological Endogenous α-Synuclein Accumulation in Oligodendrocyte Precursor Cells Potentially Induces Inclusions in Multiple System AtrophySemantic proximity Read ↗

Epsin2, a novel target for multiple system atrophy therapy via α-synuclein/FABP7 propagationSemantic proximity Read ↗

Lipid dysfunction and pathogenesis of multiple system atrophy.Semantic proximity Read ↗

Age‐related motor dysfunction and neuropathology in a transgenic mouse model of multiple system atrophySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.