Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Treatment of cholestatic fibrosis by altering gene expression of Cthrc1: Implications for autoimmune and non-autoimmune liver disease

1 Aug 2015

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Digestive system and related health
Share to Genetic factors in colorectal cancer
Share to Liver physiology and pathology

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 26238209
DOI: 10.1016/j.jaut.2015.07.010

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Treatment of cholestatic fibrosis by altering gene expression of Cthrc1: Implications for autoimmune and non-autoimmune liver diseasePMID 26238209Read ↗

Myofibroblast-specific YY1 promotes liver fibrosisSemantic proximity Read ↗

Epigenetic remodelling licences adult cholangiocytes for organoid formation and liver regenerationSemantic proximity Read ↗

Genetics and epigenetics in the fibrogenic evolution of chronic liver diseasesSemantic proximity Read ↗

Current Therapies for Cholestatic DiseasesSemantic proximity Read ↗

Sex-determining region Y gene promotes liver fibrosis and accounts for sexual dimorphism in its pathophysiologySemantic proximity Read ↗

MeCP2 controls the expression of RASAL1 in the hepatic fibrosis in ratsSemantic proximity Read ↗

Advances in precision gene editing for liver fibrosis: From technology to therapeutic applicationsSemantic proximity Read ↗

Cholangiocyte RUNX1 Orchestrates Fibrogenic and Inflammatory Signaling to Drive Biliary FibrosisSemantic proximity Read ↗

Chemokine CXCL14 acts as a potential genetic target for liver fibrosisSemantic proximity Read ↗

Hepatocyte TEAD1 drives epithelial-stromal remodeling during cholestatic liver injurySemantic proximity Read ↗

Molecular pathogenesis of cholestasis.Semantic proximity Read ↗

Pathogenesis of cholangiocarcinoma: From genetics to signalling pathwaysSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.