Phenotypic Association Analyses With Copy Number Variation in Recurrent Depressive Disorder.

doi:10.1016/j.biopsych.2015.02.025

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Phenotypic Association Analyses With Copy Number Variation in Recurrent Depressive Disorder.

Biological psychiatry - 15 Feb 2016

Rucker James J H, Tansey Katherine E, Rivera Margarita, Pinto Dalila, Cohen-Woods Sarah, Uher Rudolf, Aitchison Katherine J, Craddock Nick, Owen Michael J, Jones Lisa, Jones Ian, Korszun Ania, Barnes Michael R, Preisig Martin, Mors Ole, Maier Wolfgang, Rice John, Rietschel Marcella, Holsboer Florian, Farmer Anne E, Craig Ian W, Scherer Stephen W, McGuffin Peter, Breen Gerome

Abstract excerpt

BACKGROUND: Defining the molecular genomic basis of the likelihood of developing depressive disorder is a considerable challenge. We previously associated rare, exonic deletion copy number variants (CNV) with recurrent depressive disorder (RDD). Sex chromosome abnormalities also have been observed to co-occur with RDD. METHODS: In this reanalysis of our RDD dataset (N = 3106 cases; 459 screened control samples...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Case-Control Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chromosome Aberrations

Share to Databases, Genetic

Share to Depressive Disorder

Share to DNA Copy Number Variations

Share to Genome-Wide Association Study

Share to Linear Models

Share to Young Adult

Source

Open in PubMed Open full text

Identifiers

PMID: 25861698
DOI: 10.1016/j.biopsych.2015.02.025
PMCID: PMC4725574

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Phenotypic Association Analyses With Copy Number Variation in Recurrent Depressive Disorder.PMID 25861698Read ↗

Genome-wide association analysis of copy number variation in recurrent depressive disorder.Semantic proximity Read ↗

Association of Rare Copy Number Variants With Risk of DepressionSemantic proximity Read ↗

The role of rare copy number variants in early-onset depressionSemantic proximity Read ↗

Association between copy number variants in 16p11.2 and major depressive disorder in a German case-control sample.Semantic proximity Read ↗

Population-Based Risk of Psychiatric Disorders Associated With Recurrent Copy Number Variants.Semantic proximity Read ↗

Population-based Risk of Psychiatric Disorders Associated with Recurrent CNVsSemantic proximity Read ↗

Investigation of copy number variation in subjects with major depression based on whole-genome sequencing data.Semantic proximity Read ↗

Copy number variation and neurodevelopmental problems in females and males in the general populationSemantic proximity Read ↗

The successful search for genetic loci associated with depression.Semantic proximity Read ↗

Structural genomic variation as risk factor for idiopathic recurrent miscarriage.Semantic proximity Read ↗

Secondary analysis of GenRED data (Genetics of Recurrent Early-Onset major Depression) using MERLIN.Semantic proximity Read ↗

Sparse whole-genome sequencing identifies two loci for major depressive disorderSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.