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Article

Somatic Mutations in MLH1 and MSH2 Are a Frequent Cause of Mismatch-Repair Deficiency in Lynch Syndrome-Like Tumors

9 Dec 2013

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 24333619
DOI: 10.1053/j.gastro.2013.12.002

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Related research

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Somatic Mutations in MLH1 and MSH2 Are a Frequent Cause of Mismatch-Repair Deficiency in Lynch Syndrome-Like TumorsPMID 24333619Read ↗

Cancer Risks Associated With Germline Mutations in MLH1, MSH2, and MSH6 Genes in Lynch SyndromeSemantic proximity Read ↗

Identification of new genetic alterations in MLH1, MSH2 and MSH6 using IHC and HRM analysis in Lynch syndrome-suspected patientsSemantic proximity Read ↗

Mismatch repair genes in Lynch syndrome: a reviewSemantic proximity Read ↗

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Incidence and molecular characteristics of deficient mismatch repair conditions across nine different tumors and identification of germline variants involved in Lynch-like syndromeSemantic proximity Read ↗

New Insights into Lynch Syndrome: A Narrative Review.Semantic proximity Read ↗

Noncoding Aberrations in Mismatch Repair Genes Underlie a Substantial Part of the Missing Heritability in Lynch SyndromeSemantic proximity Read ↗

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Carcinogenesis and microsatellite instability: the interrelationship between genetics and epigeneticsSemantic proximity Read ↗

Mismatch repair genes founder mutations and cancer susceptibility in Lynch syndrome.Semantic proximity Read ↗

Mismatch repair deficiency testing in clinical practiceSemantic proximity Read ↗

Urinary Tract Cancer in Lynch Syndrome; Increased Risk in Carriers of MSH2 Mutations.Semantic proximity Read ↗

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