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Article

Replacing Shox2 with human SHOX leads to congenital disc degeneration of the temporomandibular joint in mice

18 Nov 2013

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Share to Oral and Maxillofacial Pathology
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 24248941
DOI: 10.1007/s00441-013-1743-2

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Replacing Shox2 with human SHOX leads to congenital disc degeneration of the temporomandibular joint in micePMID 24248941Read ↗

Shox2-deficiency leads to dysplasia and ankylosis of the temporomandibular joint in miceSemantic proximity Read ↗

Overexpression of Shox2 leads to congenital dysplasia of the temporomandibular joint in mice.Semantic proximity Read ↗

SHOX mutations.Semantic proximity Read ↗

<i>Shox2</i>-deficient mice exhibit a rare type of incomplete clefting of the secondary palateSemantic proximity Read ↗

A mouse model for human short-stature syndromes identifies Shox2 as an upstream regulator of Runx2 during long-bone development.Semantic proximity Read ↗

[SHOX haploinsufficiency].Semantic proximity Read ↗

SHOX defects in idiopathic short stature.Semantic proximity Read ↗

The short stature homeobox gene SHOX is involved in skeletal abnormalities in Turner syndrome.Semantic proximity Read ↗

Mice with an anterior cleft of the palate survive neonatal lethality.Semantic proximity Read ↗

SHOX duplications found in some cases with type I Mayer-Rokitansky-Kuster-Hauser syndromeSemantic proximity Read ↗

A gene desert required for regulatory control of pleiotropic <i>Shox2</i> expression and embryonic survivalSemantic proximity Read ↗

SOX9 in cartilage development and diseaseSemantic proximity Read ↗

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