Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Elucidating the Pseudomonas aeruginosa fatty acid degradation pathway: identification of additional fatty acyl-CoA synthetase homologues.

PloS one - 1 Jan 2013

Zarzycki-Siek Jan, Norris Michael H, Kang Yun, Sun Zhenxin, Bluhm Andrew P, McMillan Ian A, Hoang Tung T

Abstract excerpt

The fatty acid (FA) degradation pathway of Pseudomonas aeruginosa, an opportunistic pathogen, was recently shown to be involved in nutrient acquisition during BALB/c mouse lung infection model. The source of FA in the lung is believed to be phosphatidylcholine, the major component of lung surfact...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Coenzyme A Ligases
Share to Fatty Acids
Share to Metabolic Networks and Pathways

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Pseudomonas aeruginosa

Share to Terpenes

Share to Time Factors

Share to Virulence Factors

Source

Open in PubMed Open full text

Identifiers

PMID: 23737986
DOI: 10.1371/journal.pone.0064554
PMCID: PMC3667196

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-23737986-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Elucidating the Pseudomonas aeruginosa fatty acid degradation pathway: identification of additional fatty acyl-CoA synthetase homologues.PMID 23737986Read ↗

Multiple FadD acyl-CoA synthetases contribute to differential fatty acid degradation and virulence in Pseudomonas aeruginosa.Semantic proximity Read ↗

Blocking phosphatidylcholine utilization in Pseudomonas aeruginosa, via mutagenesis of fatty acid, glycerol and choline degradation pathways, confirms the importance of this nutrient source in vivo.Semantic proximity Read ↗

Elucidating the Functions of fabF1 and fabF2 in Pseudomonas aeruginosa: Implications for Fatty Acid Metabolism and PathogenicitySemantic proximity Read ↗

Pseudomonas aeruginosa aerobic fatty acid desaturase DesB is important for virulence factor production.Semantic proximity Read ↗

The long-chain fatty acid-CoA ligase FadD1 monitors <i>Pseudomonas aeruginosa</i> quorum sensing as a receptor of cis-2-decenoic acidSemantic proximity Read ↗

The fadXDEBA locus of Staphylococcus aureus is required for metabolism of exogenous palmitic acid and in vivo growthSemantic proximity Read ↗

PvrA is a novel regulator that contributes to Pseudomonas aeruginosa pathogenesis by controlling bacterial utilization of long chain fatty acids.Semantic proximity Read ↗

<i>Pseudomonas aeruginosa</i> core metabolism exerts a widespread growth-independent control on virulenceSemantic proximity Read ↗

A divergent Pseudomonas aeruginosa palmitoyltransferase essential for cystic fibrosis-specific lipid A.Semantic proximity Read ↗

Pseudomonas aeruginosa core metabolism exerts a widespread growth-independent control on virulence.Semantic proximity Read ↗

Virulence determinants from a cystic fibrosis isolate of Pseudomonas aeruginosa include isocitrate lyaseSemantic proximity Read ↗

Characterization of Alanine Catabolism inPseudomonas aeruginosaand Its Importance for Proliferation In VivoSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.