Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Interaction of the TaqIA polymorphism and poor parental socialization on changes in adolescent marijuana use.

Substance use & misuse - 1 Feb 2013

Vaske Jamie

Abstract excerpt

The current study uses data from the genetic subsample from the National Longitudinal Study of Adolescent Health (Add Health) in waves I and II (ages of 11-19 and 12-20 respectively) to investigate the interaction of the TaqIA polymorphism and poor parental socialization on changes in adolescent marijuana use. Results reveal that TaqIA interacts with poor parental rule setting, but not quality of mother-child...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adolescent Behavior
Share to Gene-Environment Interaction
Share to Marijuana Abuse

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Parent-Child Relations

Share to Parenting

Share to Protein Serine-Threonine Kinases

Share to Taq Polymerase

Share to Young Adult

Source

Identifiers

PMID: 23302056
DOI: 10.3109/10826084.2012.754898

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Interaction of the TaqIA polymorphism and poor parental socialization on changes in adolescent marijuana use.PMID 23302056Read ↗

Affiliation with substance-using peers: Examining gene-environment correlations among parent monitoring, polygenic risk, and children's impulsivity.Semantic proximity Read ↗

The MAOA promoter polymorphism, disruptive behavior disorders, and early onset substance use disorder: gene–environment interactionSemantic proximity Read ↗

The Interplay of MAOA and Peer Influences in Predicting Adult Criminal Behavior.Semantic proximity Read ↗

Preliminary evidence for a gene-environment interaction in predicting alcohol use disorders in adolescents.Semantic proximity Read ↗

MAOA, childhood maltreatment, and antisocial behavior: meta-analysis of a gene-environment interaction.Semantic proximity Read ↗

Parent and peer influences on emerging adult substance use disorder: A genetically informed studySemantic proximity Read ↗

Changing environmental influences on substance use across development.Semantic proximity Read ↗

Deviant socialization mediates transmissible and contextual risk on cannabis use disorder development: a prospective study.Semantic proximity Read ↗

Genotype-environment correlation by intervention effects underlying middle childhood peer rejection and associations with adolescent marijuana use.Semantic proximity Read ↗

Childhood adversity, monoamine oxidase a genotype, and risk for conduct disorder.Semantic proximity Read ↗

Genotype-environment correlations in late childhood and early adolescence: antisocial behavioral problems and coercive parenting.Semantic proximity Read ↗

Interaction between dopamine D2 receptor genotype and parental rule-setting in adolescent alcohol use: evidence for a gene-parenting interaction.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.