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Article

TLR4 inactivation protects from graft-versus-host disease after allogeneic hematopoietic stem cell transplantation

24 Dec 2012

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Share to Hematopoietic Stem Cell Transplantation
Share to Immune Cell Function and Interaction
Share to Immune Response and Inflammation

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23262974
DOI: 10.1038/cmi.2012.58

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Related research

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TLR4 inactivation protects from graft-versus-host disease after allogeneic hematopoietic stem cell transplantationPMID 23262974Read ↗

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Transplanting stem cells: Potential targets for immune attack. Modulating the immune response against embryonic stem cell transplantationSemantic proximity Read ↗

The TLR1/2 agonist PAM3CSK4 instructs commitment of human hematopoietic stem cells to a myeloid cell fateSemantic proximity Read ↗

The ex vivo toll-like receptor 7 tolerance induction in donor lymphocytes prevents murine acute graft-versus-host disease.Semantic proximity Read ↗

Regulatory Dendritic Cells Protect Mice from Murine Acute Graft-versus-Host Disease and Leukemia RelapseSemantic proximity Read ↗

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Increased susceptibility for aspergillosis and post-transplant immune deficiency in patients with gene variants of TLR4 after stem cell transplantation.Semantic proximity Read ↗

Cellular and molecular mechanisms in graft-versus-host diseaseSemantic proximity Read ↗

Host NLRP6 exacerbates graft-versus-host disease independent of gut microbial compositionSemantic proximity Read ↗

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