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Article

The genetic basis of primary aldosteronism.

Current hypertension reports - 1 Apr 2012

Funder John W

Abstract excerpt

Primary aldosteronism is commonly regarded as largely sporadic, but both germline and somatic mutations are increasingly recognized as underlying the condition. Three germline mutations causing familial hyperaldosteronism have been described, dubbed FH I (due to a CYP11B1/CYP11B2 chimera), FH II...

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Share to G Protein-Coupled Inwardly-Rectifying Potassium Channels
Share to KCNJ5 gene
Share to Genetic Predisposition to Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Germ-Line Mutation

Share to Hyperaldosteronism

Share to Nerve Tissue Proteins

Share to Potassium Channels, Tandem Pore Domain

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Source

Identifiers

PMID: 22359160
DOI: 10.1007/s11906-012-0255-x

Genes

KCNJ5

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-22359160-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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