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Article

Abnormal accumulation of human transmembrane (TMEM)-176A and 176B proteins is associated with cancer pathology

12 Jan 2012

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22244448
DOI: 10.1016/j.acthis.2011.12.006

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Related research

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Abnormal accumulation of human transmembrane (TMEM)-176A and 176B proteins is associated with cancer pathologyPMID 22244448Read ↗

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TMEM160 Promotes Tumor Growth in Lung Adenocarcinoma and Cervical Adenocarcinoma Cell LinesSemantic proximity Read ↗

Topological analysis of TMEM180, a newly identified membrane protein that is highly expressed in colorectal cancer cellsSemantic proximity Read ↗

A Pan-cancer Analysis of the Role of the Transmembrane Protein 91(TMEM91) in Human TumorsSemantic proximity Read ↗

Oncogenic roles of GPR176 in breast cancer: a potential marker of aggressiveness and a potential target of gene therapy.Semantic proximity Read ↗

Tmem176B and Tmem176A are associated with the immature state of dendritic cells.Semantic proximity Read ↗

Identification and expression of a new type II transmembrane protein in human mast cellsSemantic proximity Read ↗

The influence of a caveolin-1 mutant on the function of P-glycoprotein.Semantic proximity Read ↗

Elevated expression of TMEM205, a hypothetical membrane protein, is associated with cisplatin resistanceSemantic proximity Read ↗

Methylation of TMEM176A , a key ERK Signaling Regulator, is a Novel Synthetic Lethality Marker of ATM Inhibitors in Human Lung CancerSemantic proximity Read ↗

Missense mutations in the calcium-activated chloride channel TMEM16A promote tumor growth by activating oncogenic signaling in Human CancerSemantic proximity Read ↗

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