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Article

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome

31 May 2011

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 21625233
DOI: 10.1038/leu.2011.101

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndromePMID 21625233Read ↗

Haploinsufficiency of A20 caused by a novel pathogenic missense variant of TNFAIP3 and successfully treated with anti-TNF and immunosuppressive therapiesSemantic proximity Read ↗

Different genetic alteration of A20 in a Sézary syndrome case with Vα2-Jα22 T cell clone.Semantic proximity Read ↗

Genetic Mutations Associated With TNFAIP3 (A20) Haploinsufficiency and Their Impact on Inflammatory Diseases.Semantic proximity Read ↗

Haploinsufficiency of A20 caused by a novel nonsense variant or entire deletion of TNFAIP3 is clinically distinct from Behçet’s diseaseSemantic proximity Read ↗

A novel missense mutation in TNFAIP3 causes haploinsufficiency of A20.Semantic proximity Read ↗

Identification of Gene Mutations and Fusion Genes in Patients with Sézary Syndrome.Semantic proximity Read ↗

Multicenter international cohort study of HA20 reveals novel genetic architecture and phenotypic evolutionSemantic proximity Read ↗

Haploinsufficiency of TNFAIP3 (A20) by germline mutation is involved in autoimmune lymphoproliferative syndrome.Semantic proximity Read ↗

Multicenter international cohort study of haploinsufficiency of A20 reveals novel genetic architecture and phenotypic evolution.Semantic proximity Read ↗

Sézary Syndrome: Translating Genetic Diversity into Personalized Medicine.Semantic proximity Read ↗

Diverse transcriptomic and mutational patterns but limited functional pathway alterations in patient-derived Sézary syndrome cells.Semantic proximity Read ↗

Haploinsufficiency of A20 caused by heterozygous mutations in the TNFAIP3 gene:A Case SeriesSemantic proximity Read ↗

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