Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Extracellular matrix formation enhances the ability of Streptococcus pneumoniae to cause invasive disease.

PloS one - 1 Jan 2011

Trappetti Claudia, Ogunniyi Abiodun D, Oggioni Marco R, Paton James C

Abstract excerpt

During infection, pneumococci exist mainly in sessile biofilms rather than in planktonic form, except during sepsis. However, relatively little is known about how biofilms contribute to pneumococcal pathogenesis. Here, we carried out a biofilm assay on opaque and transparent variants of a clinical serotype 19F strain WCH159. After 4 days incubation, scanning electron microscopy revealed that opaque biofilm...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Administration, Intranasal
Share to Bacterial Adhesion
Share to Bacterial Capsules

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Biological Assay

Share to Colony Count, Microbial

Share to Extracellular Matrix

Share to Pneumococcal Infections

Share to Streptococcus pneumoniae

Source

Open in PubMed Open full text

Identifiers

PMID: 21611130
DOI: 10.1371/journal.pone.0019844
PMCID: PMC3097209

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Extracellular matrix formation enhances the ability of Streptococcus pneumoniae to cause invasive disease.PMID 21611130Read ↗

Streptococcus pneumoniae in biofilms are unable to cause invasive disease due to altered virulence determinant production.Semantic proximity Read ↗

Biofilm formation by Streptococcus pneumoniae: role of choline, extracellular DNA, and capsular polysaccharide in microbial accretion.Semantic proximity Read ↗

The choline-binding proteins PspA, PspC and LytA of <i>Streptococcus pneumoniae</i> and their role on host cellular adhesion and damageSemantic proximity Read ↗

Biofilm formation in Streptococcus pneumoniaeSemantic proximity Read ↗

Label-free microscopy enables high-throughput identification of genes controlling biofilm developmentSemantic proximity Read ↗

A soluble host signal drives rapid, brain-predominant capsular thickening in <i>Streptococcus pneumoniae</i> via a putative sodium-dependent transporter (SPD_0642) and capsular prepromoter sequenceSemantic proximity Read ↗

Isolation of Streptococcus pneumoniae biofilm mutants and their characterization during nasopharyngeal colonization.Semantic proximity Read ↗

The LuxS/AI-2 Quorum-Sensing System of Streptococcus pneumoniae Is Required to Cause Disease, and to Regulate Virulence- and Metabolism-Related Genes in a Rat Model of Middle Ear InfectionSemantic proximity Read ↗

Impaired capsular polysaccharide is relevant to enhanced biofilm formation and lower virulence in Streptococcus pneumoniae.Semantic proximity Read ↗

Function of BriC Peptide in the Pneumococcal Competence and Virulence PortfolioSemantic proximity Read ↗

Function of BriC peptide in the pneumococcal competence and virulence portfolioSemantic proximity Read ↗

Streptococcus pyogenes Biofilm Growth In Vitro and In Vivo and Its Role in Colonization, Virulence, and Genetic ExchangeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.