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Article

Follistatin and follistatin like-3 differentially regulate adiposity and glucose homeostasis.

Obesity (Silver Spring, Md.) - 1 Oct 2011

Brown Melissa L, Bonomi Lara, Ungerleider Nathan, Zina Jessica, Kimura Fuminori, Mukherjee Abir, Sidis Yisrael, Schneyer Alan

Abstract excerpt

Transforming growth factor-β superfamily ligands, including activin and myostatin, modulate body composition, islet function, and glucose homeostasis. Their bioactivity is controlled by the antagonists follistatin (FST) and FST like-3 (FSTL3). The hypothesis tested was that FST and FSTL3 have distinct roles in regulating body composition, glucose homeostasis, and islet function through regulation of activin and...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 21546932
DOI: 10.1038/oby.2011.97
PMCID: PMC3179827

Genes

FSTL3
FST

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

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