Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Apolipoprotein E genotypes modulate fibrosis progression in patients with chronic hepatitis C and persistently normal transaminases.

Journal of gastroenterology and hepatology - 1 Feb 2011

Fabris Carlo, Vandelli Carmen, Toniutto Pierluigi, Minisini Rosalba, Colletta Cosimo, Falleti Edmondo, Smirne Carlo, Pirisi Mario

Abstract excerpt

BACKGROUND AND AIM: Carriage of the apolipoprotein E (Apo E) variants, E2, E3 and E4, affects cholesterol metabolism and may be involved in the persistence of hepatitis C virus (HCV) infection. Our aim was to verify whether carriage of specific Apo E variants modulates the course of hepatitis C. METHODS: We studied a cohort of 116 HCV-positive patients (49 male subjects) with persistently normal transaminases and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alanine Transaminase
Share to Apolipoproteins E

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Biopsy

Share to Chi-Square Distribution

Share to Cholesterol

Share to Hepatitis C, Chronic

Share to Liver Cirrhosis

Source

Identifiers

PMID: 21261723
DOI: 10.1111/j.1440-1746.2010.06403.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Apolipoprotein E genotypes modulate fibrosis progression in patients with chronic hepatitis C and persistently normal transaminases.PMID 21261723Read ↗

Apolipoprotein epsilon3 allele is associated with persistent hepatitis C virus infection.Semantic proximity Read ↗

Natural history of hepatitis C virus carriers with persistently normal aminotransferase levels.Semantic proximity Read ↗

Genetic polymorphism at the apolipoprotein E locus affects the outcome of chronic hepatitis B.Semantic proximity Read ↗

Histological features and HLA class II alleles in hepatitis C virus chronically infected patients with persistently normal alanine aminotransferase levels.Semantic proximity Read ↗

Impact of apolipoprotein E genetic polymorphisms on liver disease: An essential reviewSemantic proximity Read ↗

Histological and virological features and follow-up of hepatitis C virus carriers with normal aminotransferase levels: the Italian prospective study of the asymptomatic C carriers (ISACC).Semantic proximity Read ↗

Apolipoprotein E allele frequencies in chronic and self-limited hepatitis C suggest a protective effect of APOE4 in the course of hepatitis C virus infection.Semantic proximity Read ↗

HLA-E gene polymorphisms in chronic hepatitis C: Impact on HLA-E liver expression and disease severity.Semantic proximity Read ↗

Disease progression in chronic hepatitis C: modifiable and nonmodifiable factors.Semantic proximity Read ↗

Clinical, histological, and virological features of hepatitis C virus carriers with persistently normal or abnormal alanine transaminase levels.Semantic proximity Read ↗

Carriage of the apolipoprotein E-epsilon4 allele and histologic outcome of recurrent hepatitis C after antiviral treatment.Semantic proximity Read ↗

Incidentally detected asymptomatic hepatitis C virus infection with significant fibrosis: Possible impacts on management.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.