Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The effects of dexamethasone, ascorbic acid, and β-glycerophosphate on osteoblastic differentiation by regulating estrogen receptor and osteopontin expression.

The Journal of surgical research - 1 Mar 2012

Park Jun-Beom

Abstract excerpt

BACKGROUND: Ascorbic acid (AA), β-glycerophosphate (GP), and dexamethasone (DEX) are the compounds known to favor the expression of the osteoblastic phenotype in several bone cell systems. MATERIALS AND METHODS: In this report, the combination effects of differentiation agents on osteoprecursor cells were evaluated. The effect on cell proliferation was determined by a cell viability test with morphologic...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alkaline Phosphatase
Share to Ascorbic Acid
Share to Calcification, Physiologic

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dexamethasone

Share to Glycerophosphates

Share to Models, Animal

Share to Osteoblasts

Share to Osteopontin

Source

Identifiers

PMID: 21035140
DOI: 10.1016/j.jss.2010.09.010

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The effects of dexamethasone, ascorbic acid, and β-glycerophosphate on osteoblastic differentiation by regulating estrogen receptor and osteopontin expression.PMID 21035140Read ↗

Differing responses of osteogenic cell lines to β-glycerophosphateSemantic proximity Read ↗

Osteoblast differentiation of human bone marrow cells under continuous and discontinuous treatment with dexamethasone.Semantic proximity Read ↗

Osteoblastic phenotype of rat marrow stromal cells cultured in the presence of dexamethasone, beta-glycerolphosphate, and L-ascorbic acid.Semantic proximity Read ↗

Diverse effect of BMP-2 homodimer on mesenchymal progenitors of different originSemantic proximity Read ↗

Strontium ranelate treatment of human primary osteoblasts promotes an osteocyte-like phenotype while eliciting an osteoprotegerin response.Semantic proximity Read ↗

Phenotypic effects of continuous or discontinuous treatment with dexamethasone and/or calcitriol on osteoblasts differentiated from rat bone marrow stromal cells.Semantic proximity Read ↗

Optimisation of the differing conditions required for bone formation in vitro by primary osteoblasts from mice and ratsSemantic proximity Read ↗

Dexamethasone, BMP-2, and 1,25-dihydroxyvitamin D enhance a more differentiated osteoblast phenotype: validation of an in vitro model for human bone marrow-derived primary osteoblasts.Semantic proximity Read ↗

Genistein Promotion of Osteogenic Differentiation through BMP2/SMAD5/RUNX2 SignalingSemantic proximity Read ↗

Effect of bisphosphonates on viability, proliferation, and dexamethasone-induced apoptosis of articular chondrocytesSemantic proximity Read ↗

Comparison of Fetal and Adult Marrow Stromal Cells in Osteogenesis with and without GlucocorticoidsSemantic proximity Read ↗

Differentiation of human bone marrow osteogenic stromal cells in vitro: induction of the osteoblast phenotype by dexamethasone.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.