Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic variants of glutathione S-transferases mu, theta, and pi display no susceptibility to inflammatory bowel disease in the Danish population.

Scandinavian journal of gastroenterology - 1 Sept 2010

Ernst Anja, Andersen Vibeke, Østergaard Mette, Jacobsen Bent A, Dagiliene Enrika, Pedersen Inge S, Drewes Asbjørn M, Okkels Henrik, Krarup Henrik B

Abstract excerpt

INTRODUCTION: A combination of genetic predisposition and interactions with environmental factors are believed to be responsible for disease phenotype and disease progression in inflammatory bowel diseases. The harmful effect of smoking and other environmental factors is believed to be highly dep...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Colitis, Ulcerative
Share to Crohn Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Denmark

Share to Genetic Predisposition to Disease

Share to Glutathione S-Transferase pi

Share to Glutathione Transferase

Share to Inflammatory Bowel Diseases

Share to Smoking

Source

Identifiers

PMID: 20459366
DOI: 10.3109/00365521.2010.490594

Genes

GSTM1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20459366-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic variants of glutathione S-transferases mu, theta, and pi display no susceptibility to inflammatory bowel disease in the Danish population.PMID 20459366Read ↗

Susceptibility to ulcerative colitis and Crohn's disease: interactions between glutathione S-transferase GSTM1 and GSTT1 genotypes.Semantic proximity Read ↗

GST Theta null genotype is associated with an increased risk for ulcerative colitis: a case-control study and meta-analysis of GST Mu and GST Theta polymorphisms in inflammatory bowel disease.Semantic proximity Read ↗

Association of Glutathione S-Transferase M1 null genotype with inflammatory bowel diseases: A systematic review and meta-analysis.Semantic proximity Read ↗

Glutathione S-Transferase T1 Null Genotype is Associated with Susceptibility to Inflammatory Bowel Disease.Semantic proximity Read ↗

Common polymorphisms in the microsomal epoxide hydrolase and N-acetyltransferase 2 genes in association with inflammatory bowel disease in the Danish population.Semantic proximity Read ↗

Genetic polymorphisms in metabolizing enzymes modifying the association between smoking and inflammatory bowel diseases.Semantic proximity Read ↗

Evaluation of Effect CAT -262C/T, SOD + 35A/C, GPx1 Pro197Leu Polymorphisms in Patients with IBD in the Polish Population.Semantic proximity Read ↗

The relationship between heritability and smoking habits in Crohn's disease. Italian Cooperative Study Group.Semantic proximity Read ↗

Exploring the Effects of Cigarette Smoking on Inflammatory Bowel Disease Using Mendelian RandomizationSemantic proximity Read ↗

Polymorphisms of glutathione S-transferase and methylenetetrahydrofolate reductase genes in Moldavian patients with ulcerative colitis: Genotype-phenotype correlationSemantic proximity Read ↗

Genetic polymorphisms of glutathione S-transferases are associated with ulcerative colitis in central China.Semantic proximity Read ↗

Relevance of GSTM1, GSTT1, and GSTP1 gene polymorphisms to gastric cancer susceptibility and phenotype.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.