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Article

Dystroglycan matrix receptor function in cardiac myocytes is important for limiting activity-induced myocardial damage.

Circulation research - 6 Nov 2009

Michele Daniel E, Kabaeva Zhyldyz, Davis Sarah L, Weiss Robert M, Campbell Kevin P

Abstract excerpt

RATIONALE: Genetic mutations in a number of putative glycosyltransferases lead to the loss of glycosylation of dystroglycan and loss of its laminin-binding activity in genetic forms of human muscular dystrophy. Human patients and glycosylation defective myd mice develop cardiomyopathy with loss o...

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Source

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Identifiers

PMID: 19797173
DOI: 10.1161/CIRCRESAHA.109.199489
PMCID: PMC2783339

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-19797173-2026-09-04.1. Manifest publication-graph-cohort-2026-09-04.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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