Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Endothelial–Mesenchymal Transition in Bleomycin-Induced Pulmonary Fibrosis

18 Sept 2009

Abstract excerpt

The pathological hallmark lesions in idiopathic pulmonary fibrosis are the fibroblastic foci, in which fibroblasts are thought to be involved in the tissue remodeling, matrix deposition, and cross-talk with alveolar epithelium. Recent evidence indicates that some fibroblasts in fibrosis may be derived from bone marrow progenitors as well as from epithelial cells through epithelial-mesenchymal transition. To...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
Share to Lung Cancer Diagnosis and Treatment
Share to Lung Cancer Treatments and Mutations

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 19767450
DOI: 10.1165/rcmb.2009-0031oc

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Endothelial–Mesenchymal Transition in Bleomycin-Induced Pulmonary FibrosisPMID 19767450Read ↗

Endothelial to Mesenchymal Transition (EndoMT) in the Pathogenesis of Human Fibrotic DiseasesSemantic proximity Read ↗

Fibroblasts in Kidney Fibrosis Emerge via Endothelial-to-Mesenchymal TransitionSemantic proximity Read ↗

Extracellular matrix remodeling associated with bleomycin-induced lung injury supports pericyte-to-myofibroblast transitionSemantic proximity Read ↗

Extracellular Matrix Remodeling Associated with Bleomycin-Induced Lung Injury Supports Pericyte-To-Myofibroblast TransitionSemantic proximity Read ↗

TGF-β-Induced Endothelial-Mesenchymal Transition in Fibrotic DiseasesSemantic proximity Read ↗

Endothelial Cells Recruit Macrophages and Contribute to a Fibrotic Milieu in Bleomycin Lung InjurySemantic proximity Read ↗

CREPT is required for pulmonary fibrosis induced by bleomycinSemantic proximity Read ↗

Fibroblast growth factor 2 decreases bleomycin-induced pulmonary fibrosis and inhibits fibroblast collagen production and myofibroblast differentiation.Semantic proximity Read ↗

Multiple stromal populations contribute to pulmonary fibrosis without evidence for epithelial to mesenchymal transitionSemantic proximity Read ↗

Roles of TGF-<i>β</i>Signals in Endothelial-Mesenchymal Transition during Cardiac FibrosisSemantic proximity Read ↗

The role of endothelial–mesenchymal transition in development and pathological processSemantic proximity Read ↗

Innovative approaches to the therapy of fibrosis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.