Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The Ig Domain Protein CD9P-1 Down-regulates CD81 Ability to Support Plasmodium yoelii Infection

18 Sept 2009

Abstract excerpt

Invasion of hepatocytes by Plasmodium sporozoites is a prerequisite for establishment of a malaria natural infection. The molecular mechanisms underlying sporozoite invasion are largely unknown. We have previously reported that CD81 is required on hepatocytes for infection by Plasmodium falciparum and Plasmodium yoelii sporozoites. CD81 belongs to the tetraspanin superfamily of transmembrane proteins. By...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Complement system in diseases
Share to Drug Transport and Resistance Mechanisms
Share to Malaria Research and Control

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 19762465
DOI: 10.1074/jbc.m109.057927

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The Ig Domain Protein CD9P-1 Down-regulates CD81 Ability to Support Plasmodium yoelii InfectionPMID 19762465Read ↗

Expression of human CD81 differently affects host cell susceptibility to malaria sporozoites depending on the Plasmodium speciesSemantic proximity Read ↗

Plasmodium sporozoites can invade hepatocytic cells independently of the Ephrin receptor A2Semantic proximity Read ↗

Plasmodium berghei EXP-1 interacts with host Apolipoprotein H during Plasmodium liver-stage developmentSemantic proximity Read ↗

Plasmodium falciparum exoerythrocytic forms require the PTEX translocon for development in human hepatocytesSemantic proximity Read ↗

Contrasting Inducible Knockdown of the Auxiliary PTEX Component PTEX88 in P. falciparum and P. berghei Unmasks a Role in Parasite VirulenceSemantic proximity Read ↗

Plasmodium falciparum exploits CD44 as a coreceptor for erythrocyte invasionSemantic proximity Read ↗

Rhoptry neck protein 11 has crucial roles during malaria parasite sporozoite invasion of salivary glands and hepatocytesSemantic proximity Read ↗

EWI-2 Is a Major CD9 and CD81 Partner and Member of a Novel Ig Protein SubfamilySemantic proximity Read ↗

Phenotype and function of MAIT cells in patients with alveolar echinococcosis.Semantic proximity Read ↗

A Plasmodium homolog of ER tubule‐forming proteins is required for parasite virulenceSemantic proximity Read ↗

<i>Plasmodium</i> liver stage developmental arrest by depletion of a protein at the parasite–host interfaceSemantic proximity Read ↗

The vaccine candidate Liver Stage Antigen 3 is exported during <i>Plasmodium falciparum</i> infection and required for liver-stage developmentSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.