Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glucose Promotes Stress Resistance in the Fungal Pathogen<i>Candida albicans</i>

17 Sept 2009

Abstract excerpt

Metabolic adaptation, and in particular the modulation of carbon assimilatory pathways during disease progression, is thought to contribute to the pathogenicity of Candida albicans. Therefore, we have examined the global impact of glucose upon the C. albicans transcriptome, testing the sensitivity of this pathogen to wide-ranging glucose levels (0.01, 0.1, and 1.0%). We show that, like Saccharomyces cerevisiae,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antifungal resistance and susceptibility
Share to Plant-Microbe Interactions and Immunity
Share to Plant Pathogens and Fungal Diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 19759180
DOI: 10.1091/mbc.e09-01-0002

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glucose Promotes Stress Resistance in the Fungal Pathogen<i>Candida albicans</i>PMID 19759180Read ↗

Glucose-enhanced oxidative stress resistance—A protective anticipatory response that enhances the fitness of Candida albicans during systemic infectionSemantic proximity Read ↗

Stress adaptation in a pathogenic fungusSemantic proximity Read ↗

Integrative Model of Oxidative Stress Adaptation in the Fungal Pathogen Candida albicans.Semantic proximity Read ↗

Transcriptional profiling reveals the role of Candida albicans Rap1 in oxidative stress responseSemantic proximity Read ↗

Identifying infection-associated genes of<i>Candida albicans</i>in the postgenomic eraSemantic proximity Read ↗

An integrated systems biology approach establishes arginine biosynthesis as a metabolic weakness in <i>Candida albicans</i> during host infectionSemantic proximity Read ↗

Carbon source-induced reprogramming of the cell wall proteome and secretome modulates the adherence and drug resistance of the fungal pathogen Candida albicans.Semantic proximity Read ↗

<i>Candida albicans</i> cell wall glycosidases <i>DFG5</i> and <i>DCW1</i> are required for biofilm formation and Hog-1 signalingSemantic proximity Read ↗

Carbon substrates promotes stress resistance and drug tolerance in clinical isolates of Candida tropicalisSemantic proximity Read ↗

A conserved stress-activated protein kinase regulates a core stress response in the human pathogen Candida albicans.Semantic proximity Read ↗

MIG1Regulates Resistance of Candida albicans against the Fungistatic Effect of Weak Organic AcidsSemantic proximity Read ↗

Stress- and metabolic responses of <i>Candida albicans</i> require Tor1 kinase N-terminal HEAT repeatsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.