Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Salmonella translocates across an in vitro M cell model independently of SPI-1 and SPI-2

1 Dec 2008

Abstract excerpt

We have used an in vitro model of intestinal M cells to examine the mechanisms by which Salmonella enterica translocates across these specialized cells, which constitute a primary site of infection of the mammalian host. S. enterica can invade cultured cells by deploying a type III secretion system (TTSS) encoded within Salmonella pathogenicity island 1 (SPI-1) to translocate effector proteins into the host cell...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to RNA Interference and Gene Delivery
Share to Salmonella and Campylobacter epidemiology
Share to Viral gastroenteritis research and epidemiology

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 19047755
DOI: 10.1099/mic.0.2008/021162-0

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 24 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Salmonella translocates across an in vitro M cell model independently of SPI-1 and SPI-2PMID 19047755Read ↗

Translocation of enteropathogenic Escherichia coli across an in vitro M cell model is regulated by its type III secretion system.Semantic proximity Read ↗

Analysis of interactions of Salmonella type three secretion mutants with 3-D intestinal epithelial cells.Semantic proximity Read ↗

SPI2 T3SS effectors facilitate enterocyte apical to basolateral transmigration of Salmonella -containing vacuoles in vivoSemantic proximity Read ↗

Salmonella typhimurium transcytoses flagellin via an SPI2-mediated vesicular transport pathway.Semantic proximity Read ↗

Salmonella enterica serovar Typhimurium SPI-1 and SPI-2 shape the transcriptional landscape of epithelial cells in a human intestinal organoid model systemSemantic proximity Read ↗

Cross-Talk Between the Intestinal Epithelium and Salmonella TyphimuriumSemantic proximity Read ↗

Salmonella effector driven invasion of the gut epithelium: breaking in and setting the house on fireSemantic proximity Read ↗

From vacuole to cytosol – Disruptive invasion triggers cytosolic release of <i>Salmonella</i> Paratyphi A and subsequent cytosolic motility favors evasion of xenophagySemantic proximity Read ↗

Minimal SPI1-T3SS effector requirement for Salmonella enterocyte invasion and intracellular proliferation in vivoSemantic proximity Read ↗

Functions of the Salmonella pathogenicity island 2 (SPI-2) type III secretion system effectorsSemantic proximity Read ↗

Functions and effectors of the Salmonella pathogenicity island 2 type III secretion system.Semantic proximity Read ↗

Salmonella pathogenicity island 1 differentially modulates bacterial entry to dendritic and non‐phagocytic cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.