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Article

sodA is essential for virulence of Borrelia burgdorferi in the murine model of Lyme disease.

Molecular microbiology - 1 Feb 2009

Esteve-Gassent Maria D, Elliott Nathaniel L, Seshu J

Abstract excerpt

Borrelia burgdorferi, the causative agent of Lyme disease, has a limited set of genes to combat oxidative/nitrosative stress encountered in its tick vector or mammalian hosts. We inactivated the gene encoding for superoxide dismutase A (sodA, bb0153), an enzyme mediating the dismutation of superoxide anions and examined the in vitro and in vivo phenotype of the mutant. There were no significant differences in the...

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Identifiers

PMID: 19040638
DOI: 10.1111/j.1365-2958.2008.06549.x

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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