Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Malignant transformation-related genes in meningiomas: allelic loss on 1p36 and methylation status of p73 and RASSF1A

1 Aug 2007

Abstract excerpt

OBJECT: Analysis of meningiomas supports the suggestion that loss of heterozygosity (LOH) of chromosome arm 1p plays an important role in malignancy. The aim of this study was to identify genes related to meningioma progression from the benign state to the atypical and anaplastic states by examining 1p LOH and the promoter methylation of RASSF1A and p73. METHODS: The authors studied 40 surgical specimens (22 WHO...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Meningioma and schwannoma management
Share to Neurofibromatosis and Schwannoma Cases
Share to Pituitary Gland Disorders and Treatments

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 17695396
DOI: 10.3171/jns-07/08/0398

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 23 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Malignant transformation-related genes in meningiomas: allelic loss on 1p36 and methylation status of p73 and RASSF1APMID 17695396Read ↗

A consistent region of deletion on 1p36 in meningiomas: identification and relation to malignant progression.Semantic proximity Read ↗

Genetic Alterations Associated With Progression and Recurrence in MeningiomasSemantic proximity Read ↗

DNA methylation of multiple promoter-associated CpG islands in meningiomas: relationship with the allelic status at 1p and 22q.Semantic proximity Read ↗

Allelic losses on chromosomes 14, 10, and 1 in atypical and malignant meningiomas: a genetic model of meningioma progression.Semantic proximity Read ↗

Loss of material from chromosome arm 1p during malignant progression of meningioma revealed by fluorescent in Situ hybridizationSemantic proximity Read ↗

Genomic Profiling of Atypical Meningiomas Associates Gain of 1q With Poor Clinical OutcomeSemantic proximity Read ↗

Identification of a consistent region of allelic loss on 1p32 in meningiomas: correlation with increased morbidity.Semantic proximity Read ↗

Loss of heterozygosity analysis of benign, atypical, and anaplastic meningiomas.Semantic proximity Read ↗

Pathology and Molecular Genetics of Meningioma: Recent AdvancesSemantic proximity Read ↗

Molecular Biological Determinations of Meningioma Progression and RecurrenceSemantic proximity Read ↗

Epigenetic subclassification of meningiomas based on genome-wide DNA methylation analysesSemantic proximity Read ↗

DNA Methylation in the Malignant Transformation of MeningiomasSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.