Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

IL-1alpha, IL-1ra and IL-8 are differentially induced by Candida in experimental oral candidiasis.

Oral diseases - 1 Jul 2007

Jayatilake J A M S, Samaranayake L P, Lu Q, Jin L J

Abstract excerpt

OBJECTIVE: To investigate the expression of interleukin-1alpha (IL-1alpha), IL-1ra and IL-8 by the oral epithelium challenged by various Candida species. MATERIALS AND METHODS: In vitro candidiasis was induced by C. albicans wild type SC5314, its EFG1, CPH1 and secretory aspartyl proteinase (SAP)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Candida
Share to Candida albicans
Share to Candida tropicalis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Candidiasis, Oral

Share to Hyphae

Share to Interleukin-1alpha

Share to Interleukin-8

Share to Mouth Mucosa

Source

Identifiers

PMID: 17577331
DOI: 10.1111/j.1601-0825.2007.01318.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17577331-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

IL-1alpha, IL-1ra and IL-8 are differentially induced by Candida in experimental oral candidiasis.PMID 17577331Read ↗

Hyphal invasion of Candida albicans inhibits the expression of human beta-defensins in experimental oral candidiasis.Semantic proximity Read ↗

Candida albicans Pathogenicity and Epithelial ImmunitySemantic proximity Read ↗

Invasive Phenotype ofCandida albicansAffects the Host Proinflammatory Response to InfectionSemantic proximity Read ↗

EGR1 regulates oral epithelial cell responses to Candida albicans via the EGFR- ERK1/2 pathwaySemantic proximity Read ↗

Virulence and pathogenicity of<i>Candida albicans</i>is enhanced in biofilms containing oral bacteriaSemantic proximity Read ↗

In vivo transcript profiling of Candida albicans identifies a gene essential for interepithelial disseminationSemantic proximity Read ↗

A Biphasic Innate Immune MAPK Response Discriminates between the Yeast and Hyphal Forms of Candida albicans in Epithelial CellsSemantic proximity Read ↗

Real-time PCR expression profiling of genes encoding potential virulence factors in Candida albicans biofilms: identification of model-dependent and -independent gene expressionSemantic proximity Read ↗

Invasion of Candida albicans correlates with expression of secreted aspartic proteinases during experimental infection of human epidermis.Semantic proximity Read ↗

Adaptations of Candida albicans for Growth in the Mammalian Intestinal TractSemantic proximity Read ↗

Quantitative expression of the Candida albicans secreted aspartyl proteinase gene family in human oral and vaginal candidiasisSemantic proximity Read ↗

Potential role of Candida albicans secreted aspartic protease 9 in serum induced-hyphal formation and interaction with oral epithelial cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.