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Article

Association study of tardive dyskinesia and twelve DRD2 polymorphisms in schizophrenia patients.

The international journal of neuropsychopharmacology - 1 Oct 2007

Zai Clement C, Hwang Rudi W, De Luca Vincenzo, Müller Daniel J, King Nicole, Zai Gwyneth C, Remington Gary, Meltzer Herbert Y, Lieberman Jeffrey A, Potkin Steven G, Kennedy James L

Abstract excerpt

Tardive dyskinesia (TD) is a side-effect of chronic antipsychotic medication. Abnormalities in dopaminergic activity in the nigrostriatal system have been most often suggested to be involved because the agents which cause TD share in common potent antagonism of dopamine D2 receptors (DRD2), that notably is not balanced by effects such as more potent serotonin (5-HT)2A antagonism. Thus, a number of studies have...

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Share to Antipsychotic Agents
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chi-Square Distribution

Share to Dyskinesia, Drug-Induced

Share to Linkage Disequilibrium

Share to Receptors, Dopamine D2

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Source

Identifiers

PMID: 16959057
DOI: 10.1017/S1461145706007152

Genes

DRD2
DRD3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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