Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Epigenetic Regulation of Varicella-Zoster Virus Open Reading Frames 62 and 63 in Latently Infected Human Trigeminal Ganglia

26 Apr 2006

Abstract excerpt

Open reading frames (ORFs) 21, 29, 62, 63, and 66 of varicella-zoster virus (VZV) are transcribed during latency in human ganglia. ORF 63 is the most frequently expressed gene, and ORF 62 encodes a transcriptional activator. The mechanisms regulating the expression of these genes are not well understood, although analyses of other alphaherpesviruses indicate a role for chromatin in virus gene regulation during...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytomegalovirus and herpesvirus research
Share to Herpesvirus Infections and Treatments
Share to HIV Research and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 16641283
DOI: 10.1128/jvi.80.10.4921-4926.2006

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Epigenetic Regulation of Varicella-Zoster Virus Open Reading Frames 62 and 63 in Latently Infected Human Trigeminal GangliaPMID 16641283Read ↗

Varicella-Zoster Virus Gene 66 Transcription and Translation in Latently Infected Human GangliaSemantic proximity Read ↗

Repression of varicella zoster virus gene expression during quiescent infection in the absence of detectable histone depositionSemantic proximity Read ↗

Repression of varicella zoster virus gene expression during quiescent infection in the absence of detectable histone depositionSemantic proximity Read ↗

Varicella-zoster virus (VZV) transcription during latency in human ganglia: detection of transcripts mapping to genes 21, 29, 62, and 63 in a cDNA library enriched for VZV RNASemantic proximity Read ↗

Expression of protein encoded by varicella-zoster virus open reading frame 63 in latently infected human ganglionic neurons.Semantic proximity Read ↗

Varicella-zoster virus VLT-ORF63 fusion transcript induces broad viral gene expression during reactivation from neuronal latencySemantic proximity Read ↗

Varicella-Zoster Virus Immediate-Early 63 Protein Interacts with Human Antisilencing Function 1 Protein and Alters Its Ability To Bind Histones H3.1 and H3.3Semantic proximity Read ↗

Expression of varicella-zoster virus VLT-ORF63 fusion transcript induces broad viral gene expression during reactivation from neuronal latencySemantic proximity Read ↗

Prevalence and Abundance of Latently Transcribed Varicella-Zoster Virus Genes in Human GangliaSemantic proximity Read ↗

The varicella-zoster virus immediate-early 62 promoter contains a negative regulatory element that binds transcriptional factor NF-Y.Semantic proximity Read ↗

Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone AxisSemantic proximity Read ↗

Varicella-zoster virus gene 63 encodes an immediate-early protein that is abundantly expressed during latencySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.