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Article

The E23K polymorphism in Kir6.2 gene and coronary heart disease.

Clinica chimica acta; international journal of clinical chemistry - 1 May 2006

Xiong Chenling, Zheng Fang, Wan Jun, Zhou Xin, Yin Zhinong, Sun Xiaobo

Abstract excerpt

BACKGROUND: The G to A mutation in the Kir 6.2, the ATP-sensitive potassium channel subunit, resulted a glutamate (E) to lysine (K) substitution at codon 23, and the A allele was shown to have a relationship with high risk to type 2 diabetes in previous study. Their role in coronary heart disease (CHD) has not been evaluated. We hypothesized that the polymorphism would be associated with increased susceptibility...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Potassium Channels, Inwardly Rectifying

Source

Identifiers

PMID: 16455067
DOI: 10.1016/j.cca.2005.11.032

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

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