Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Adipokines and the insulin resistance syndrome in familial partial lipodystrophy caused by a mutation in lamin A/C.

Diabetologia - 1 Dec 2005

Wong S P Y, Huda M, English P, Bargiota A, Wilding J P H, Johnson A, Corrall R, Pinkney J H

Abstract excerpt

AIMS/HYPOTHESIS: Familial partial lipodystrophy (FPLD) and obesity are both associated with increased risks of type 2 diabetes and cardiovascular disease. Although adipokines have been implicated, few data exist in subjects with FPLD; therefore we investigated a family with FPLD due to a lamin A/C mutation in order to determine how abnormalities of the plasma adipokine profile relate to insulin resistance and the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adiponectin
Share to Insulin Resistance

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Interleukin-1

Share to Interleukin-6

Share to Laminin

Share to Leptin

Share to Metabolic Syndrome

Share to Resistin

Source

Identifiers

PMID: 16320084
DOI: 10.1007/s00125-005-0038-x

Genes

TNF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Adipokines and the insulin resistance syndrome in familial partial lipodystrophy caused by a mutation in lamin A/C.PMID 16320084Read ↗

Characterisation of non-obese diabetic patients with marked insulin resistance identifies a novel familial partial lipodystrophy-associated PPARγ mutation (Y151C).Semantic proximity Read ↗

Familial partial lipodystrophy: a monogenic form of the insulin resistance syndrome.Semantic proximity Read ↗

Lipodystrophic syndromes: From diagnosis to treatment.Semantic proximity Read ↗

Familial partial lipodystrophy syndromes.Semantic proximity Read ↗

Elevated serum C-reactive protein and free fatty acids among nondiabetic carriers of missense mutations in the gene encoding lamin A/C (LMNA) with partial lipodystrophy.Semantic proximity Read ↗

Association between nuclear lamin A/C R482Q mutation and partial lipodystrophy with hyperinsulinemia, dyslipidemia, hypertension, and diabetes.Semantic proximity Read ↗

[Mutations of the lamin A/C gene causing familial partial lipodystrophy: significance for the development of hyperlipidemia and insulin resistant diabetes mellitus].Semantic proximity Read ↗

[Adipokine genetics: unbalanced protein secretion by human adipose tissue as a cause of the metabolic syndrome].Semantic proximity Read ↗

Lipodystrophy: metabolic insights from a rare disorderSemantic proximity Read ↗

Monogenic forms of insulin resistance: apertures that expose the common metabolic syndrome.Semantic proximity Read ↗

[Lipodystrophies].Semantic proximity Read ↗

The molecular basis of genetic lipodystrophies.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.