Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enhanced Na-Li countertransport: a marker of inherited susceptibility to type 2 diabetes.

International journal of epidemiology - 1 Oct 2005

Vaccaro Olga, Cuomo Vincenzo, Trevisan Maurizio, Cirillo Massimo, Panarelli Walter, Laurenzi Martino, Mancini Mario, Riccardi Gabriele

Abstract excerpt

UNLABELLED: Introduction The association between type 2 diabetes and hypertension has long been described, but the mechanisms remain unclear. Na-Li countertransport (Na-Li CT) activity is viewed as a marker of inherited pre-disposition to hypertension, especially if associated with other metabolic abnormalities. Aim To evaluate whether enhanced Na-Li CT activity is a predictor of type 2 diabetes. METHODS: Study...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antiporters
Share to Blood Glucose

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Blood Pressure

Share to Body Mass Index

Share to Cholesterol, HDL

Share to Incidence

Share to Lithium

Share to Sodium

Source

Identifiers

PMID: 16087689
DOI: 10.1093/ije/dyi160

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enhanced Na-Li countertransport: a marker of inherited susceptibility to type 2 diabetes.PMID 16087689Read ↗

Genetic predisposition to hypertension (as detected by Na+/Li+ countertransport) and risk of diabetic nephropathy in childhood diabetes.Semantic proximity Read ↗

Role of thiamine status and genetic variability in transketolase and other pentose phosphate cycle enzymes in the progression of diabetic nephropathy.Semantic proximity Read ↗

Association of Urinary Sodium Excretion and Left Ventricular Hypertrophy in Patients With Type 2 Diabetes: A Cross-Section StudySemantic proximity Read ↗

Increased red cell sodium-lithium countertransport and lymphocyte cytosolic calcium are separate phenotypes in patients with essential hypertension.Semantic proximity Read ↗

Genetic and physiological association of diabetes susceptibility with raised Na+/H+ exchange activity.Semantic proximity Read ↗

Apolipoprotein E gene polymorphism is related to metabolic abnormalities, but does not influence erythrocyte membrane lipid composition or sodium-lithium countertransport activity in essential hypertension.Semantic proximity Read ↗

Abnormalities in insulin sensitivity, vascular resistance and erythrocyte cation transport are independent genetic traits in familial hypertension.Semantic proximity Read ↗

The apolipoprotein E2 allele modulates activity and maximal velocity of the sodium-lithium countertransporter.Semantic proximity Read ↗

Role of apolipoprotein C‐III overproduction in diabetic dyslipidaemiaSemantic proximity Read ↗

Angiotensin-converting enzyme (ACE) gene polymorphism and the erythrocyte sodium-lithium countertransporter (SLC) phenotype in hypertension.Semantic proximity Read ↗

Insulin increases sodium (Na+) channel density in A6 epithelia: implications for expression of hypertension.Semantic proximity Read ↗

Renal glucose excretion as a function of blood glucose concentration in subjects with type 2 diabetes--results of a hyperglycaemic glucose clamp study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.