Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic polymorphisms of oxidative and antioxidant enzymes and arsenic-related hypertension.

Journal of toxicology and environmental health. Part A - 1 Sept 2005

Hsueh Yu-Mei, Lin Pinpin, Chen Hui-Wen, Shiue Horng-Sheng, Chung Chi-Jung, Tsai Chiao-Tzu, Huang Yung-Kay, Chiou Hung-Yi, Chen Chien-Jen

Abstract excerpt

The association of 4 genetic polymorphisms, NAD(P)H oxidase, manganesesuperoxide dismutase (MnSOD), catalase, and endothelial nitric oxide synthase (e-NOS), was assessed with arsenic-related hypertension risk among 79 hypertensive cases and 213 controls in an arseniasis-hyperendemic area of Taiwan. Overall, MnSOD polymorphism significantly increased the risk of hypertension regardless of arsenic exposure. NADPH...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arsenic
Share to Catalase

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Environmental Exposure

Share to NADPH Oxidases

Share to Nitric Oxide Synthase

Share to Nitric Oxide Synthase Type III

Share to Superoxide Dismutase

Share to Water Supply

Source

Identifiers

PMID: 16076760
DOI: 10.1080/15287390590967414

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic polymorphisms of oxidative and antioxidant enzymes and arsenic-related hypertension.PMID 16076760Read ↗

Elevated risk of hypertension induced by arsenic exposure in Taiwanese rural residents: possible effects of manganese superoxide dismutase (MnSOD) and 8-oxoguanine DNA glycosylase (OGG1) genes.Semantic proximity Read ↗

Association of arsenic-induced cardiovascular disease susceptibility with genetic polymorphisms.Semantic proximity Read ↗

Essential hypertension in patients exposed to high-arsenic exposed areas in western China: Genetic susceptibility and urinary arsenic metabolism characteristics.Semantic proximity Read ↗

Genetic Polymorphisms in Arsenic Metabolism and Oxidative Stress Genes Linked to Increased Cardiovascular Disease Risk in Arsenic Exposed Bangladeshi PopulationSemantic proximity Read ↗

Arsenic methylation, GSTO1 polymorphisms, and metabolic syndrome in an arseniasis endemic area of southwestern Taiwan.Semantic proximity Read ↗

Significantly increased risk of carotid atherosclerosis with arsenic exposure and polymorphisms in arsenic metabolism genes.Semantic proximity Read ↗

Synergistic effect of polymorphisms of paraoxonase gene cluster and arsenic exposure on electrocardiogram abnormality.Semantic proximity Read ↗

Research for type 2 diabetes mellitus in endemic arsenism areas in central China: role of low level of arsenic exposure and KEAP1 rs11545829 polymorphism.Semantic proximity Read ↗

Effect of gene-environment interaction (arsenic exposure - PON1 Q192R polymorphism) on cardiovascular disease biomarkers in Mexican population.Semantic proximity Read ↗

Polymorphic variations in manganese superoxide dismutase (MnSOD) and endothelial nitric oxide synthase (eNOS) genes contribute to the development of type 2 diabetes mellitus in the Chinese Han population.Semantic proximity Read ↗

Risk of carotid atherosclerosis associated with genetic polymorphisms of apolipoprotein E and inflammatory genes among arsenic exposed residents in Taiwan.Semantic proximity Read ↗

Urinary arsenic metabolism, genetic susceptibility, and their interaction on type 2 diabetes.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.