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Article

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

24 Apr 2005

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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PMID: 15852005
DOI: 10.1038/ng1552

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Related research

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Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathwaysPMID 15852005Read ↗

Inversin, Wnt signaling and primary ciliaSemantic proximity Read ↗

Inversin relays Frizzled-8 signals to promote proximal pronephros developmentSemantic proximity Read ↗

Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination.Semantic proximity Read ↗

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Biallelic ANKS6 mutations cause late-onset ciliopathy with chronic kidney disease through YAP dysregulation.Semantic proximity Read ↗

ANKS6 is the critical activator of NEK8 kinase in embryonic situs determination and organ patterning.Semantic proximity Read ↗

Inversin forms a complex with catenins and N-cadherin in polarized epithelial cells.Semantic proximity Read ↗

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Anks3 interacts with nephronophthisis proteins and is required for normal renal developmentSemantic proximity Read ↗

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Nicastrin/miR-30a-3p/RAB31 Axis Regulates Keratinocyte Differentiation by Impairing EGFR Signaling in Familial Acne Inversa.Semantic proximity Read ↗

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