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Article

Anks3 interacts with nephronophthisis proteins and is required for normal renal development

11 Feb 2015

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Share to Fetal and Pediatric Neurological Disorders
Share to Genetic and Kidney Cyst Diseases
Share to Renal and related cancers

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 25671767
DOI: 10.1038/ki.2015.17

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Related research

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Anks3 interacts with nephronophthisis proteins and is required for normal renal developmentPMID 25671767Read ↗

Akirin proteins in development and disease: critical roles and mechanisms of actionSemantic proximity Read ↗

Mutations in ANKS6 cause a nephronophthisis-like phenotype with ESRD.Semantic proximity Read ↗

PAX2 in human kidney malformations and diseaseSemantic proximity Read ↗

Nephrin – a unique structural and signaling protein of the kidney filterSemantic proximity Read ↗

Biallelic ANKS6 mutations cause late-onset ciliopathy with chronic kidney disease through YAP dysregulation.Semantic proximity Read ↗

Nephrin Localizes at the Podocyte Filtration Slit Area and Is Characteristically Spliced in the Human KidneySemantic proximity Read ↗

The Inv compartment of renal cilia is an intraciliary signal-activating center to phosphorylate ANKS6.Semantic proximity Read ↗

The ANKS3/BICC1 protein complex is a master post-transcriptional regulator of <i>NPHP1</i> ciliopathy-gene transcriptsSemantic proximity Read ↗

ANKS6 is a central component of a nephronophthisis module linking NEK8 to INVS and NPHP3.Semantic proximity Read ↗

ANKS3 Co-Localises with ANKS6 in Mouse Renal Cilia and Is Associated with Vasopressin Signaling and Apoptosis In Vivo in Mice.Semantic proximity Read ↗

Essential roles of Sall1 in kidney developmentSemantic proximity Read ↗

Human ankyrins and their contribution to disease biology: An updateSemantic proximity Read ↗

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