Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Components of the peptidoglycan-recycling pathway modulate invasion and intracellular survival of Salmonella enterica serovar Typhimurium.

Cellular microbiology - 1 Jan 2005

Folkesson Anders, Eriksson Sofia, Andersson Mats, Park James T, Normark Staffan

Abstract excerpt

beta-Lactam resistance in enteric bacteria is frequently caused by mutations in ampD encoding a cytosolic N-acetylmuramyl- l-alanine amidase. Such mutants are blocked in murein (peptidoglycan) recycling and accumulate cytoplasmic muropeptides that interact with the transcriptional activator ampR, which de-represses beta-lactamase expression. Salmonella enterica serovar Typhimurium, an extensively studied enteric...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Wall
Share to Colony Count, Microbial

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to N-Acetylmuramoyl-L-alanine Amidase

Share to Nitric Oxide

Share to Peptidoglycan

Share to Salmonella Infections

Share to Salmonella typhimurium

Share to Spleen

Source

Identifiers

PMID: 15617530
DOI: 10.1111/j.1462-5822.2004.00443.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Components of the peptidoglycan-recycling pathway modulate invasion and intracellular survival of Salmonella enterica serovar Typhimurium.PMID 15617530Read ↗

A Peptidoglycan Amidase Activator Impacts Salmonella enterica Serovar Typhimurium Gut Infection.Semantic proximity Read ↗

Mutations in ampG or ampD affect peptidoglycan fragment release from Neisseria gonorrhoeae.Semantic proximity Read ↗

Temporal and anatomical host resistance to chronic Salmonella infection is quantitatively dictated by Nramp1 and influenced by host genetic background.Semantic proximity Read ↗

Cytosolic intermediates for cell wall biosynthesis and degradation control inducible beta-lactam resistance in gram-negative bacteria.Semantic proximity Read ↗

Identification and genetic characterization of PmrA-regulated genes and genes involved in polymyxin B resistance in Salmonella enterica serovar typhimurium.Semantic proximity Read ↗

An alternative route for recycling of N-acetylglucosamine from peptidoglycan involves the N-acetylglucosamine phosphotransferase system in Escherichia coli.Semantic proximity Read ↗

Mutations of the Listeria monocytogenes peptidoglycan N-deacetylase and O-acetylase result in enhanced lysozyme sensitivity, bacteriolysis, and hyperinduction of innate immune pathways.Semantic proximity Read ↗

Salmonella typhimurium mutants that downregulate phagocyte nitric oxide production.Semantic proximity Read ↗

MurA escape mutations uncouple peptidoglycan biosynthesis from PrkA signaling.Semantic proximity Read ↗

Distinct innate responses are induced by attenuated Salmonella enterica serovar Typhimurium mutants.Semantic proximity Read ↗

MurA escape mutations uncouple peptidoglycan biosynthesis from PrkA signalingSemantic proximity Read ↗

Identification and functional analysis of Salmonella enterica serovar Typhimurium PmrA-regulated genes.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.