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Article

Promoter Hypermethylation Frequency and BRAF Mutations Distinguish Hereditary Non-Polyposis Colon Cancer from Sporadic MSI-H Colon Cancer

1 Jun 2004

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Share to Colorectal Cancer Screening and Detection
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Share to Genetic factors in colorectal cancer

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15340260
DOI: 10.1023/b:fame.0000039861.30651.c8

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Related research

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Promoter Hypermethylation Frequency and BRAF Mutations Distinguish Hereditary Non-Polyposis Colon Cancer from Sporadic MSI-H Colon CancerPMID 15340260Read ↗

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Frequent alterations of the beta-catenin and TCF-4 genes, but not of the APC gene, in colon cancers with high-frequency microsatellite instability.Semantic proximity Read ↗

Patients with colorectal cancer associated with Lynch syndrome and MLH1 promoter hypermethylation have similar prognosesSemantic proximity Read ↗

Aberrant DNA methylation in hereditary nonpolyposis colorectal cancer without mismatch repair deficiency.Semantic proximity Read ↗

Mutations in both KRAS and BRAF may contribute to the methylator phenotype in colon cancer.Semantic proximity Read ↗

Concomitant RASSF1A hypermethylation and KRAS/BRAF mutations occur preferentially in MSI sporadic colorectal cancer.Semantic proximity Read ↗

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