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Article

In vivo expression of podocyte slit diaphragm-associated proteins in nephrotic patients with NPHS2 mutation

11 Aug 2004

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Share to Complement system in diseases
Share to Renal Diseases and Glomerulopathies
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15327385
DOI: 10.1111/j.1523-1755.2004.00840.x

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Related research

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In vivo expression of podocyte slit diaphragm-associated proteins in nephrotic patients with NPHS2 mutationPMID 15327385Read ↗

Genetic kidney diseases disclose the pathogenesis of proteinuria.Semantic proximity Read ↗

Genetics of nephrotic syndrome: connecting molecular genetics to podocyte physiologySemantic proximity Read ↗

A novel NPHS2 mutation (c.865A > G) identified in a Chinese family with steroid-resistant nephrotic syndrome alters subcellular localization of nephrin.Semantic proximity Read ↗

A novel domain regulating degradation of the glomerular slit diaphragm protein podocin in cell culture systems.Semantic proximity Read ↗

Disease-causing missense mutations in NPHS2 gene alter normal nephrin trafficking to the plasma membraneSemantic proximity Read ↗

Nephrotic plasma alters slit diaphragm-dependent signaling and translocates nephrin, Podocin, and CD2 associated protein in cultured human podocytes.Semantic proximity Read ↗

New developments in steroid-resistant nephrotic syndrome.Semantic proximity Read ↗

Nephrin Localizes at the Podocyte Filtration Slit Area and Is Characteristically Spliced in the Human KidneySemantic proximity Read ↗

Nephrin TRAP mice lack slit diaphragms and show fibrotic glomeruli and cystic tubular lesions.Semantic proximity Read ↗

Genes and Podocytes â€“ New Insights into Mechanisms of PodocytopathySemantic proximity Read ↗

Nephrin-signature molecule of the glomerular podocyte?Semantic proximity Read ↗

Nephrotic Syndrome With Mutations in NPHS2: The Role of R229Q and Implications for Genetic CounselingSemantic proximity Read ↗

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